Literature DB >> 32896088

Neural Correlates of Motor Imagery of Gait in Amyotrophic Lateral Sclerosis.

Malek Abidi1, Giovanni de Marco1,2, Fatma Grami1, Nicolas Termoz1,2, Annabelle Couillandre1,2, Giorgia Querin3,4, Peter Bede3,4,5, Pierre-Francois Pradat3,4,6.   

Abstract

BACKGROUND: Gait impairment is poorly characterized in amyotrophic lateral sclerosis (ALS), despite increasing evidence of extrapyramidal and cerebellar dysfunction. Gait impairment adds to the considerable motor disability of ALS patients and requires targeted multidisciplinary interventions.
PURPOSE: To assess gait imagery-specific networks and functional adaptation in ALS. STUDY TYPE: Prospective. POPULATION: Seventeen ALS patients with lower motor neuron predominant (LMNp) disability, 14 patients with upper motor neurons predominant (UMNp) disease, and 14 healthy controls were included. FIELD STRENGTH/SEQUENCES: 3T / gradient echo echo planar (GE-EPI). ASSESSMENT: Subjects performed a dual motor imagery task: normal and precision gait. The Movement Imagery Questionnaire - Revised Second Version (MIQ-rs) was used to appraise movement imagery in each participant. Study group-specific activation patterns were evaluated during motor imagery of gait. Additional generalized psychophysiological interaction analyses were carried out using the supplementary motor area, caudate, cerebellum, and superior parietal lobule as seed regions. STATISTICAL TESTS: Repeated-measures analysis of variance (ANOVA) was used to compare time imagery and MIQ-rs scores between groups. Size effects were also reported as partial eta squared (η2). One-way ANOVA was performed to explore differences in terms of connexions during motor imagery tasks.
RESULTS: A significant increase in imagery time in UMNp patients compared to controls (P < 0.05) and LMNp (P < 0.05) during imagined gait was demonstrated. UMNp patients exhibited altered supplementary motor area, precentral gyrus, superior parietal lobule, and dorsolateral prefrontal cortex activation and increased orbitofrontal (pFDR(False Discovery Rate)  < 0.05), posterior parietal (pFDR < 0.05) caudate (pFDR < 0.05), and cerebellar (pFDR < 0.05) signal during imagined locomotion. Increased effective connectivity of the striato-cerebellar and parieto-cerebellar circuits was also demonstrated. Additional activation was detected in the insula and cingulate cortex. DATA
CONCLUSION: Enhanced striato- and parieto-cerebellar networks in UMNp ALS patients are likely to represent a compensatory response to impaired postural control. LEVEL OF EVIDENCE: 2 TECHNICAL EFFICACY STAGE: 5.
© 2020 International Society for Magnetic Resonance in Medicine.

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Mesh:

Year:  2020        PMID: 32896088     DOI: 10.1002/jmri.27335

Source DB:  PubMed          Journal:  J Magn Reson Imaging        ISSN: 1053-1807            Impact factor:   4.813


  9 in total

Review 1.  Cerebellar pathology in motor neuron disease: neuroplasticity and neurodegeneration.

Authors:  Rangariroyashe H Chipika; Grainne Mulkerrin; Pierre-François Pradat; Aizuri Murad; Fabrice Ango; Cédric Raoul; Peter Bede
Journal:  Neural Regen Res       Date:  2022-11       Impact factor: 6.058

Review 2.  Motor Imagery: How to Assess, Improve Its Performance, and Apply It for Psychosis Diagnostics.

Authors:  Alla Chepurova; Alexander Hramov; Semen Kurkin
Journal:  Diagnostics (Basel)       Date:  2022-04-11

3.  Pathological neural networks and artificial neural networks in ALS: diagnostic classification based on pathognomonic neuroimaging features.

Authors:  Peter Bede; Aizuri Murad; Orla Hardiman
Journal:  J Neurol       Date:  2021-09-28       Impact factor: 6.682

4.  Infratentorial pathology in frontotemporal dementia: cerebellar grey and white matter alterations in FTD phenotypes.

Authors:  Mary Clare McKenna; Rangariroyashe H Chipika; Stacey Li Hi Shing; Foteini Christidi; Jasmin Lope; Mark A Doherty; Jennifer C Hengeveld; Alice Vajda; Russell L McLaughlin; Orla Hardiman; Siobhan Hutchinson; Peter Bede
Journal:  J Neurol       Date:  2021-05-13       Impact factor: 6.682

5.  White matter microstructure alterations in frontotemporal dementia: Phenotype-associated signatures and single-subject interpretation.

Authors:  Mary Clare McKenna; Marlene Tahedl; Aizuri Murad; Jasmin Lope; Orla Hardiman; Siobhan Hutchinson; Peter Bede
Journal:  Brain Behav       Date:  2022-01-24       Impact factor: 2.708

Review 6.  The strength of corticomotoneuronal drive underlies ALS split phenotypes and reflects early upper motor neuron dysfunction.

Authors:  Andrew Eisen; Peter Bede
Journal:  Brain Behav       Date:  2021-10-28       Impact factor: 2.708

7.  Mapping cortical disease-burden at individual-level in frontotemporal dementia: implications for clinical care and pharmacological trials.

Authors:  Mary Clare McKenna; Marlene Tahedl; Jasmin Lope; Rangariroyashe H Chipika; Stacey Li Hi Shing; Mark A Doherty; Jennifer C Hengeveld; Alice Vajda; Russell L McLaughlin; Orla Hardiman; Siobhan Hutchinson; Peter Bede
Journal:  Brain Imaging Behav       Date:  2021-12-09       Impact factor: 3.224

8.  Amyotrophic lateral sclerosis and cerebellum.

Authors:  Renata Kabiljo; Alfredo Iacoangeli; Ammar Al-Chalabi; Ivana Rosenzweig
Journal:  Sci Rep       Date:  2022-07-22       Impact factor: 4.996

9.  Clusters of anatomical disease-burden patterns in ALS: a data-driven approach confirms radiological subtypes.

Authors:  Peter Bede; Aizuri Murad; Jasmin Lope; Orla Hardiman; Kai Ming Chang
Journal:  J Neurol       Date:  2022-03-25       Impact factor: 6.682

  9 in total

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