Literature DB >> 32870458

High serum levels of TAC and early mortality in patients with spontaneous intracerebral haemorrhage.

Leonardo Lorente1, María M Martín2, Antonia Pérez-Cejas3, Agustín F González-Rivero3, Rafael Sabatel4, Luis Ramos-Gómez5, Mónica Argueso6, Jordi Solé-Violán7, Juan J Cáceres8, Alejandro Jiménez9, Victor García-Marín10.   

Abstract

OBJECTIVE: Oxidation contributes to secondary brain injury after spontaneous intracerebral haemorrhage (SIH). One study found lower levels of total antioxidant capacity (TAC) in the blood in patients with SIH than in healthy subjects. However, there are no data on blood TAC levels and survival in patients with SIH. Therefore, the objective of our study was to determine if an association exists between serum TAC levels and mortality in patients with SIH.
METHODS: We included patients with severe supratentorial SIH. We considered severe when Glasgow Coma Scale (GCS) < 9. Patients from 6 Spanish hospitals were included in this observational and prospective study. Serum TAC levels at days 1, 4 and 8 of SIH were determined. Thirty-day mortality was our end-point study.
RESULTS: Non-surviving patients compared with surviving patients showed higher serum TAC levels at day 1 (p < 0.001), 4 (p < 0.001) and 8 (p = 0.001). An area under the curve was found for the prediction of 30-day mortality by serum TAC levels of 0.92 (95% CI = 0.85-96%; p < 0.001). Multiple logistic regression analysis showed an association of serum TAC levels with 30-day mortality (odds ratio = 16.513; 95% CI = 2.548-107.015; p = 0.003) controlling for midline shift, glycemia, early evacuation of SIH, intracerebral haemorrhage (ICH) score, age and volume of SIH.
CONCLUSIONS: The new findings of this study are that serum TAC levels are higher in non-surviving than in surviving patients, and that they are associated with mortality and could be used to predict mortality.

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Keywords:  Apoptosis; Mortality; Patients; Spontaneous intracerebral haemorrhage; Total antioxidant capacity

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Year:  2020        PMID: 32870458     DOI: 10.1007/s10072-020-04692-z

Source DB:  PubMed          Journal:  Neurol Sci        ISSN: 1590-1874            Impact factor:   3.307


  1 in total

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Journal:  Exp Ther Med       Date:  2019-11-21       Impact factor: 2.447

  1 in total
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Journal:  ASN Neuro       Date:  2021 Jan-Dec       Impact factor: 4.146

  1 in total

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