Literature DB >> 32846335

Ivacaftor improves lung disease in patients with advanced CF carrying CFTR mutations that confer residual function.

Donatello Salvatore1, Vito Terlizzi2, Michela Francalanci2, Giovanni Taccetti2, Barbara Messore3, Carlotta Biglia3, Giovanna Pisi4, Maria Adelaide Calderazzo5, Mimma Caloiero5, Giovanna Pizzamiglio6, Fabio Majo7, Federico Cresta8, Giuseppina Leonetti9, Domenica De Venuto9.   

Abstract

BACKGROUND: Ivacaftor is an innovative treatment for CF. Ivacaftor monotherapy in a phase III trial for patients with F508del and a residual function (RF) mutation showed improvement in lung function. We evaluated the effectiveness and safety of ivacaftor in patients with severe CF carrying RF mutations.
METHODS: Data were collected from Italian CF centers with patients enrolled in an ivacaftor compassionate use program. Data were collected 1 year before and 1 year after commencement of ivacaftor.
RESULTS: Twenty-six patients received ivacaftor. The mean [standard deviation (SD)] percent predicted FEV1 significantly increased from 33.9% (8.3) before treatment to 44.0% (10.7) after 12 months of treatment (p < 0.00001). The mean distance in the 6-min walking-test significantly improved from 458.2 (110.5) m at baseline to 524.8 (91.9) m after 12 months (p < 0.00001). The overall number of days of antibiotic therapy decreased from 1693 during the year before ivacaftor to 714 in the year following ivacaftor, and the number of days of intravenous antibiotic treatment dropped from 714 to 88; both results were statistically significant (p < 0.00001). Patients needing intravenous therapy decreased from 23 to 5 of 26. The mean (SD) sweat chloride level decreased from a baseline of 79 (22.3) mmol/L to 65 (30.6) mmol/L, but this variation was not significant (p = 0.26). No safety concerns were registered.
CONCLUSIONS: In patients with CFTR mutations that confer RF with severe lung disease, treatment with Ivacaftor is safe and results in a clinically significant improvement that was evident at 1 month and maintained at 12 months.
Copyright © 2020 Elsevier Ltd. All rights reserved.

Entities:  

Keywords:  CFTR; Cystic fibrosis; Ivacaftor; Residual function

Mesh:

Substances:

Year:  2020        PMID: 32846335     DOI: 10.1016/j.rmed.2020.106073

Source DB:  PubMed          Journal:  Respir Med        ISSN: 0954-6111            Impact factor:   3.415


  9 in total

1.  Pancreatic Insufficiency in a Child with p.Gly542* and c.2657+5G>A Heterozygote CFTR: A Case Report.

Authors:  Fernanda Dos Anjos; Jonatha Wruck; Thiago Inácio Teixeira do Carmo; Victor Emanuel Miranda Soares; Débora Tavares de Resende E Silva; Margarete Dulce Bagatini; Sarah Franco Vieira de Oliveira Maciel
Journal:  Clin Med Res       Date:  2022-02-07

2.  Effectiveness of Elexacaftor/Tezacaftor/Ivacaftor Therapy in Three Subjects with the Cystic Fibrosis Genotype Phe508del/Unknown and Advanced Lung Disease.

Authors:  Vito Terlizzi; Carmela Colangelo; Giovanni Marsicovetere; Michele D'Andria; Michela Francalanci; Diletta Innocenti; Eleonora Masi; Angelo Avarello; Giovanni Taccetti; Felice Amato; Marika Comegna; Giuseppe Castaldo; Donatello Salvatore
Journal:  Genes (Basel)       Date:  2021-07-29       Impact factor: 4.096

3.  Elexacaftor/Tezacaftor/Ivacaftor in Patients with Cystic Fibrosis Homozygous for the F508del Mutation and Advanced Lung Disease: A 48-Week Observational Study.

Authors:  Vincenzo Carnovale; Paola Iacotucci; Vito Terlizzi; Carmela Colangelo; Lorenza Ferrillo; Angela Pepe; Michela Francalanci; Giovanni Taccetti; Serena Buonaurio; Assunta Celardo; Laura Salvadori; Giovanni Marsicovetere; Michele D'Andria; Nicola Ferrara; Donatello Salvatore
Journal:  J Clin Med       Date:  2022-02-16       Impact factor: 4.241

4.  CRMS/CFSPID Subjects Carrying D1152H CFTR Variant: Can the Second Variant Be a Predictor of Disease Development?

Authors:  Vito Terlizzi; Rita Padoan; Laura Claut; Carla Colombo; Benedetta Fabrizzi; Marco Lucarelli; Sabina Maria Bruno; Alice Castaldo; Paolo Bonomi; Giovanni Taccetti; Antonella Tosco
Journal:  Diagnostics (Basel)       Date:  2020-12-12

5.  Ex vivo model predicted in vivo efficacy of CFTR modulator therapy in a child with rare genotype.

Authors:  Vito Terlizzi; Felice Amato; Chiara Castellani; Beatrice Ferrari; Luis J V Galietta; Giuseppe Castaldo; Giovanni Taccetti
Journal:  Mol Genet Genomic Med       Date:  2021-03-13       Impact factor: 2.183

6.  Anticipating New Treatments for Cystic Fibrosis: A Global Survey of Researchers.

Authors:  Bernardo Cabral; Vito Terlizzi; Onofrio Laselva; Carlos Conte Filho; Fabio Mota
Journal:  J Clin Med       Date:  2022-02-26       Impact factor: 4.241

Review 7.  New Therapies to Correct the Cystic Fibrosis Basic Defect.

Authors:  Christelle Bergeron; André M Cantin
Journal:  Int J Mol Sci       Date:  2021-06-08       Impact factor: 5.923

8.  Frequency of CFTR variants in southern Brazil and indication for modulators therapy in patients with cystic fibrosis.

Authors:  Eliandra da Silveira Lima; Luíse Sgarabotto Pezzin; Ana Carolina Fensterseifer; Leonardo Araújo Pinto
Journal:  Genet Mol Biol       Date:  2021-12-06       Impact factor: 1.771

9.  Hypertonic saline in people with cystic fibrosis: review of comparative studies and clinical practice.

Authors:  Vito Terlizzi; Eleonora Masi; Michela Francalanci; Giovanni Taccetti; Diletta Innocenti
Journal:  Ital J Pediatr       Date:  2021-08-06       Impact factor: 2.638

  9 in total

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