Literature DB >> 32841353

Influence of Obesity in the miRNome: miR-4454, a Key Regulator of Insulin Response Via Splicing Modulation in Prostate.

Vicente Herrero-Aguayo1,2,3,4, Juan M Jiménez-Vacas1,2,3,4, Prudencio Sáez-Martínez1,2,3,4, Enrique Gómez-Gómez1,3,5, Juan L López-Cánovas1,2,3,4, Lourdes Garrido-Sánchez4,6, Aura D Herrera-Martínez1,3,7, Laura García-Bermejo8, Manuel Macías-González4,6, José López-Miranda1,3,9, Justo P Castaño1,2,3,4, Manuel D Gahete1,2,3,4, Raúl M Luque1,2,3,4.   

Abstract

CONTEXT: Obesity is a major health problem associated with severe comorbidities, including type 2 diabetes and cancer, wherein microRNAs (miRNAs) might be useful as diagnostic/prognostic tools or therapeutic targets.
OBJECTIVE: To explore the differential expression pattern of miRNAs in obesity and their putative role in obesity-related comorbidities such as insulin resistance.
METHODS: An Affymetrix-miRNA array was performed in plasma samples from normoweight (n = 4/body mass index < 25) and obese subjects (n = 4/body mass index > 30). The main changes were validated in 2 independent cohorts (n = 221/n = 18). Additionally, in silico approaches were performed and in vitro assays applied in tissue samples and prostate (RWPE-1) and liver (HepG2) cell-lines.
RESULTS: A total of 26 microRNAs were altered (P < 0.01) in plasma of obese subjects compared to controls using the Affymetrix-miRNA array. Validation in ampler cohorts revealed that miR-4454 levels were consistently higher in obesity, associated with insulin-resistance (Homeostatic Model Assessment of Insulin Resistance/insulin) and modulated by medical (metformin/statins) and surgical (bariatric surgery) strategies. miR-4454 was highly expressed in prostate and liver tissues and its expression was increased in prostate and liver cells by insulin. In vitro, overexpression of miR-4454 in prostate cells resulted in decreased expression levels of INSR, GLUT4, and phosphorylation of AMPK/AKT/ERK, as well as in altered expression of key spliceosome components (ESRP1/ESRP2/RBM45/RNU2) and insulin-receptor splicing variants.
CONCLUSIONS: Obesity was associated to an alteration of the plasmatic miRNA landscape, wherein miR-4454 levels were higher, associated with insulin-resistance and modulated by obesity-controlling interventions. Insulin regulated miR-4454, which, in turn may impair the cellular response to insulin, in a cell type-dependent manner (i.e., prostate gland), by modulating the splicing process.
© The Author(s) 2020. Published by Oxford University Press on behalf of the Endocrine Society. All rights reserved. For permissions, please e-mail: journals.permissions@oup.com.

Entities:  

Keywords:  insulin; insulin receptor; miRNA; obesity; splicing

Year:  2021        PMID: 32841353     DOI: 10.1210/clinem/dgaa580

Source DB:  PubMed          Journal:  J Clin Endocrinol Metab        ISSN: 0021-972X            Impact factor:   5.958


  5 in total

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4.  Dysregulation of the miRNome unveils a crosstalk between obesity and prostate cancer: miR-107 asa personalized diagnostic and therapeutic tool.

Authors:  Vicente Herrero-Aguayo; Prudencio Sáez-Martínez; Juan M Jiménez-Vacas; M Trinidad Moreno-Montilla; Antonio J Montero-Hidalgo; Jesús M Pérez-Gómez; Juan L López-Canovas; Francisco Porcel-Pastrana; Julia Carrasco-Valiente; Francisco J Anglada; Enrique Gómez-Gómez; Elena M Yubero-Serrano; Alejandro Ibañez-Costa; Aura D Herrera-Martínez; André Sarmento-Cabral; Manuel D Gahete; Raúl M Luque
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