Literature DB >> 32775181

Predicting transition from oral pre-malignancy to malignancy via Bcl-2 immuno-expression: Evidence and lacunae.

Ruby Dwivedi1, Shaleen Chandra2, Divya Mehrotra1, Vineet Raj3, Rahul Pandey4.   

Abstract

Bcl-2 (B cell Lymphoma -2) family comprises of both anti-apoptotic and pro-apoptotic proteins whose altered expression or change in ratio inhibits apoptosis, and promotes tumor progression. The aim of this study is to assess the usefulness of Bcl-2 in distinguishing dysplastic or malignant epithelium from non-dysplastic or normal epithelium to aid in prediction of malignant transformation potential. MATERIAL AND
METHOD: Study group comprised of 30 cases of clinically diagnosed leukoplakia (OPMD), 15 cases of Oral Squamous Cell Carcinoma (OSCC) and 5 normal tissue samples. The labeling index of Bcl-2 was analyzed in immunohistochemically stained sections. Different statistical tools were used to analyze the data and to compare Bcl-2 expression qualitatively and quantitatively among all the groups.
RESULTS: An increasing trend of Bcl-2 immunoexpression was observed from normal epithelium to non-dysplastic and from non-dysplastic to dysplastic lesions. In OSCC, the peripheral cells in the differentiating epithelial islands (within the connective tissue) showed Bcl-2 immuno-reactivity, which gradually decreased towards the center. In contrast, intense and diffuse Bcl-2 immuno-reactivity was seen in poorly differentiated carcinoma. But the overall Bcl-2 positivity was less in OSCC as compared to dysplastic lesions.
CONCLUSION: Increased expression of Bcl-2 oncoprotein in sequentially progressing epithelial dysplasia and down-regulation in differentiating carcinoma (well and moderately differentiating OSCC) unveils the clinical relevance of Bcl-2 in early stages of OSCC tumorigenesis. The heterogenous expression of Bcl-2 in carcinoma with different grades of differentiation renders them unable to be used as an independent tool for predicting transition from oral pre-malignancy to malignancy.
© 2020 Craniofacial Research Foundation. Published by Elsevier B.V. All rights reserved.

Entities:  

Keywords:  Bcl-2; Dysplasia; Immunohistochemistry; Oral cancer

Year:  2020        PMID: 32775181      PMCID: PMC7394759          DOI: 10.1016/j.jobcr.2020.07.003

Source DB:  PubMed          Journal:  J Oral Biol Craniofac Res        ISSN: 2212-4268


  48 in total

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  1 in total

1.  Expression and bioinformatics analyses show HSP70 complements BCL2 action in oral carcinogenesis.

Authors:  Geeta Singh; Rahul Pandey; Ekta R Anthony; Shaleen Chandra; Divya Mehrotra
Journal:  J Oral Biol Craniofac Res       Date:  2022-07-20
  1 in total

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