Literature DB >> 32696951

A generalizable data-driven multicellular model of pancreatic ductal adenocarcinoma.

Boris Aguilar1, David L Gibbs1, David J Reiss2, Mark McConnell2, Samuel A Danziger2, Andrew Dervan2, Matthew Trotter3, Douglas Bassett2, Robert Hershberg4, Alexander V Ratushny2, Ilya Shmulevich1.   

Abstract

BACKGROUND: Mechanistic models, when combined with pertinent data, can improve our knowledge regarding important molecular and cellular mechanisms found in cancer. These models make the prediction of tissue-level response to drug treatment possible, which can lead to new therapies and improved patient outcomes. Here we present a data-driven multiscale modeling framework to study molecular interactions between cancer, stromal, and immune cells found in the tumor microenvironment. We also develop methods to use molecular data available in The Cancer Genome Atlas to generate sample-specific models of cancer.
RESULTS: By combining published models of different cells relevant to pancreatic ductal adenocarcinoma (PDAC), we built an agent-based model of the multicellular pancreatic tumor microenvironment, formally describing cell type-specific molecular interactions and cytokine-mediated cell-cell communications. We used an ensemble-based modeling approach to systematically explore how variations in the tumor microenvironment affect the viability of cancer cells. The results suggest that the autocrine loop involving EGF signaling is a key interaction modulator between pancreatic cancer and stellate cells. EGF is also found to be associated with previously described subtypes of PDAC. Moreover, the model allows a systematic exploration of the effect of possible therapeutic perturbations; our simulations suggest that reducing bFGF secretion by stellate cells will have, on average, a positive impact on cancer apoptosis.
CONCLUSIONS: The developed framework allows model-driven hypotheses to be generated regarding therapeutically relevant PDAC states with potential molecular and cellular drivers indicating specific intervention strategies.
© The Author(s) 2020. Published by Oxford University Press.

Entities:  

Keywords:  cancer modeling; data-driven model; multicellular model; pancreatic ductal adenocarcinoma

Year:  2020        PMID: 32696951      PMCID: PMC7374045          DOI: 10.1093/gigascience/giaa075

Source DB:  PubMed          Journal:  Gigascience        ISSN: 2047-217X            Impact factor:   6.524


  69 in total

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Journal:  Nature       Date:  2016-02-24       Impact factor: 49.962

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5.  The activated stroma index is a novel and independent prognostic marker in pancreatic ductal adenocarcinoma.

Authors:  Mert Erkan; Christoph W Michalski; Simon Rieder; Carolin Reiser-Erkan; Ivane Abiatari; Armin Kolb; Nathalia A Giese; Irene Esposito; Helmut Friess; Jörg Kleeff
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Journal:  Front Immunol       Date:  2014-10-07       Impact factor: 7.561

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Journal:  BMC Bioinformatics       Date:  2018-12-21       Impact factor: 3.169

9.  Integrative network modeling reveals mechanisms underlying T cell exhaustion.

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10.  PhysiBoSS: a multi-scale agent-based modelling framework integrating physical dimension and cell signalling.

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Journal:  Bioinformatics       Date:  2019-04-01       Impact factor: 6.937

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1.  circRNA circ_102049 Implicates in Pancreatic Ductal Adenocarcinoma Progression through Activating CD80 by Targeting miR-455-3p.

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  1 in total

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