Literature DB >> 32654539

Association of Factor V Leiden With Subsequent Atherothrombotic Events: A GENIUS-CHD Study of Individual Participant Data.

Lars Wallentin1, Folkert W Asselbergs2,3,4, Riyaz S Patel3,5, Bakhtawar K Mahmoodi6,7, Vinicius Tragante2, Marcus E Kleber8, Michael V Holmes9,10, Amand F Schmidt2,3, Raymond O McCubrey11, Laurence J Howe3, Kenan Direk3, Hooman Allayee12, Ekaterina V Baranova13, Peter S Braund14,15, Graciela E Delgado8, Niclas Eriksson1, Crystel M Gijsberts16, Yan Gong17, Jaana Hartiala12,18, Mahyar Heydarpour19,20, Gerard Pasterkamp21, Salma Kotti22, Pekka Kuukasjärvi23, Petra A Lenzini24, Daniel Levin25, Leo-Pekka Lyytikäinen26,27, Jochen D Muehlschlegel19,20, Christopher P Nelson16,14, Kjell Nikus28,29, Anna P Pilbrow30, W H Wilson Tang31,32, Sander W van der Laan33, Jessica van Setten2, Ragnar O Vilmundarson34, John Deanfield3, Panos Deloukas35, Frank Dudbridge36, Stefan James1,37, Ify R Mordi25, Andrej Teren38,39, Thomas O Bergmeijer6, Simon C Body40, Michiel Bots41,42, Ralph Burkhardt39, Rhonda M Cooper-DeHoff17,43, Sharon Cresci24,44, Nicolas Danchin45, Robert N Doughty46, Diederick E Grobbee41, Emil Hagström37,47, Stanley L Hazen31,48, Claes Held1,37, Imo E Hoefer49, G Kees Hovingh50, Julie A Johnson17,43, Marcin P Kaczor51, Mika Kähönen52,53, Olaf H Klungel13, Jari O Laurikka23,54, Terho Lehtimäki26,27, Anke H Maitland-van der Zee13,55, Ruth McPherson56, Colin N Palmer57, Adriaan O Kraaijeveld2, Carl J Pepine43, Marek Sanak51, Naveed Sattar58, Markus Scholz39,59, Tabassome Simon60,61, John A Spertus62,63, Alexandre F R Stewart34,56, Wojciech Szczeklik51, Joachim Thiery39,64, Frank L J Visseren65, Johannes Waltenberger66, A Mark Richards30,67, Chim C Lang25, Vicky A Cameron30, Axel Åkerblom1,37, Guillaume Pare68,69, Winfried März8,70,71, Nilesh J Samani14,15, Aroon D Hingorani3, Jurriën M Ten Berg6.   

Abstract

BACKGROUND: Studies examining the role of factor V Leiden among patients at higher risk of atherothrombotic events, such as those with established coronary heart disease (CHD), are lacking. Given that coagulation is involved in the thrombus formation stage on atherosclerotic plaque rupture, we hypothesized that factor V Leiden may be a stronger risk factor for atherothrombotic events in patients with established CHD.
METHODS: We performed an individual-level meta-analysis including 25 prospective studies (18 cohorts, 3 case-cohorts, 4 randomized trials) from the GENIUS-CHD (Genetics of Subsequent Coronary Heart Disease) consortium involving patients with established CHD at baseline. Participating studies genotyped factor V Leiden status and shared risk estimates for the outcomes of interest using a centrally developed statistical code with harmonized definitions across studies. Cox proportional hazards regression models were used to obtain age- and sex-adjusted estimates. The obtained estimates were pooled using fixed-effect meta-analysis. The primary outcome was composite of myocardial infarction and CHD death. Secondary outcomes included any stroke, ischemic stroke, coronary revascularization, cardiovascular mortality, and all-cause mortality.
RESULTS: The studies included 69 681 individuals of whom 3190 (4.6%) were either heterozygous or homozygous (n=47) carriers of factor V Leiden. Median follow-up per study ranged from 1.0 to 10.6 years. A total of 20 studies with 61 147 participants and 6849 events contributed to analyses of the primary outcome. Factor V Leiden was not associated with the combined outcome of myocardial infarction and CHD death (hazard ratio, 1.03 [95% CI, 0.92-1.16]; I2=28%; P-heterogeneity=0.12). Subgroup analysis according to baseline characteristics or strata of traditional cardiovascular risk factors did not show relevant differences. Similarly, risk estimates for the secondary outcomes including stroke, coronary revascularization, cardiovascular mortality, and all-cause mortality were also close to identity.
CONCLUSIONS: Factor V Leiden was not associated with increased risk of subsequent atherothrombotic events and mortality in high-risk participants with established and treated CHD. Routine assessment of factor V Leiden status is unlikely to improve atherothrombotic events risk stratification in this population.

Entities:  

Keywords:  coronary artery disease; genetic association studies; myocardial infarction; prognosis; secondary prevention; single nucleotide polymorphism; thrombosis

Mesh:

Substances:

Year:  2020        PMID: 32654539      PMCID: PMC7493828          DOI: 10.1161/CIRCULATIONAHA.119.045526

Source DB:  PubMed          Journal:  Circulation        ISSN: 0009-7322            Impact factor:   29.690


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