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Literature DB >> 32606246

Sir2 takes affirmative action to ensure equal opportunity in replication origin licensing.

Armelle Lengronne¹, Philippe Pasero².

Abstract

Entities: Gene

Mesh：

Substances：

Year: 2020 PMID： 32606246 PMCID： PMC7382207 DOI： 10.1073/pnas.2010001117

Source DB: PubMed Journal: Proc Natl Acad Sci U S A ISSN： 0027-8424 Impact factor: 11.205

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25 in total

1. Single-molecule analysis reveals clustering and epigenetic regulation of replication origins at the yeast rDNA locus.

Authors: Philippe Pasero; Aaron Bensimon; Etienne Schwob
Journal: Genes Dev Date: 2002-10-01 Impact factor: 11.361

2. The NAD(+)-dependent Sir2p histone deacetylase is a negative regulator of chromosomal DNA replication.

Authors: Donald L Pappas; Ryan Frisch; Michael Weinreich
Journal: Genes Dev Date: 2004-04-01 Impact factor: 11.361

3. Excess MCM proteins protect human cells from replicative stress by licensing backup origins of replication.

Authors: Arkaitz Ibarra; Etienne Schwob; Juan Méndez
Journal: Proc Natl Acad Sci U S A Date: 2008-06-25 Impact factor: 11.205

4. SIR2 suppresses replication gaps and genome instability by balancing replication between repetitive and unique sequences.

Authors: Eric J Foss; Uyen Lao; Emily Dalrymple; Robin L Adrianse; Taylor Loe; Antonio Bedalov
Journal: Proc Natl Acad Sci U S A Date: 2017-01-03 Impact factor: 11.205

Sir2 takes affirmative action to ensure equal opportunity in replication origin licensing.

1. Single-molecule analysis reveals clustering and epigenetic regulation of replication origins at the yeast rDNA locus.

2. The NAD(+)-dependent Sir2p histone deacetylase is a negative regulator of chromosomal DNA replication.

3. Excess MCM proteins protect human cells from replicative stress by licensing backup origins of replication.

4. SIR2 suppresses replication gaps and genome instability by balancing replication between repetitive and unique sequences.

5. Pervasive transcription fine-tunes replication origin activity.

6. Limiting replication initiation factors execute the temporal programme of origin firing in budding yeast.

Review 7. Signaling pathways of replication stress in yeast.

8. Relicensing of transcriptionally inactivated replication origins in budding yeast.

9. Mcm2, but not RPA, is a component of the mammalian early G1-phase prereplication complex.

10. Noncoding transcription influences the replication initiation program through chromatin regulation.