Literature DB >> 32562683

Effect of dulaglutide on cognitive impairment in type 2 diabetes: an exploratory analysis of the REWIND trial.

Tali Cukierman-Yaffe1, Hertzel C Gerstein2, Helen M Colhoun3, Rafael Diaz4, Luis-Emilio García-Pérez5, Mark Lakshmanan5, Angelyn Bethel5, Denis Xavier6, Jeffrey Probstfield7, Matthew C Riddle8, Lars Rydén9, Charles Messan Atisso5, Stephanie Hall10, Purnima Rao-Melacini10, Jan Basile11, William C Cushman12, Edward Franek13, Matyas Keltai14, Fernando Lanas15, Lawrence A Leiter16, Patricio Lopez-Jaramillo17, Valdis Pirags18, Nana Pogosova19, Peter J Raubenheimer20, Jonathan E Shaw21, Wayne H-H Sheu22, Theodora Temelkova-Kurktschiev23.   

Abstract

BACKGROUND: Diabetes is an independent risk factor for cognitive impairment. We aimed to investigate the association between the glucagon-like peptide-1 (GLP-1) receptor agonist dulaglutide and cognitive impairment as an exploratory analysis within the Researching Cardiovascular Events With a Weekly Incretin in Diabetes (REWIND) trial.
METHODS: REWIND is a randomised, double-blind placebo-controlled trial at 371 sites in 24 countries. We included men and women (aged ≥50 years) with either established or newly diagnosed type 2 diabetes and additional cardiovascular risk factors, glycated haemoglobin of up to 9·5% (80 mmol/mol) on a maximum of two oral glucose-lowering drugs with or without basal insulin, and a body-mass index of at least 23 kg/m2. Participants were randomly assigned (1:1) subcutaneous injections once a week of either dulaglutide (1·5 mg) or an equal volume of matching placebo. Randomisation was done using a computer-generated code with stratification by site. Participants and all study personnel were masked to treatment allocation until the database was locked. Participants were followed up at least every 6 months for the composite primary outcome of stroke, myocardial infarction, or death from cardiovascular or unknown causes. Cognitive function was assessed at baseline and during follow-up using the Montreal Cognitive Assessment (MoCA) and Digit Symbol Substitution Test (DSST). We present here the exploratory primary cognitive outcome, which was the first occurrence of a follow-up score on MoCA or DSST that was 1·5 SDs or more below the baseline mean score in the participant's country. All analyses were done using an intention-to-treat approach. The REWIND trial is registered with ClinicalTrials.gov, NCT01394952.
FINDINGS: Between Aug 18, 2011, and Aug 14, 2013, 9901 participants were randomly assigned to either dulaglutide (n=4949) or placebo (n=4952). During median follow-up of 5·4 (IQR 5·1-5·9) years, 8828 participants provided a baseline and one or more follow-up MoCA or DSST scores, of whom 4456 were assigned dulaglutide and 4372 were assigned placebo. The cognitive outcome occurred in 4·05 per 100 patient-years in participants assigned dulaglutide and 4·35 per 100 patient-years in people assigned placebo (hazard ratio [HR] 0·93, 95% CI 0·85-1·02; p=0·11). After post-hoc adjustment for individual standardised baseline scores, the hazard of substantive cognitive impairment was reduced by 14% in those assigned dulaglutide (HR 0·86, 95% CI 0·79-0·95; p=0·0018).
INTERPRETATION: Long-term treatment with dulaglutide might reduce cognitive impairment in people with type 2 diabetes. Further studies of this drug focused on brain health and cognitive function are clearly indicated. FUNDING: Eli Lilly and Company.
Copyright © 2020 Elsevier Ltd. All rights reserved.

Entities:  

Year:  2020        PMID: 32562683     DOI: 10.1016/S1474-4422(20)30173-3

Source DB:  PubMed          Journal:  Lancet Neurol        ISSN: 1474-4422            Impact factor:   44.182


  34 in total

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Review 2.  Breakdown of the blood-brain barrier: A mediator of increased Alzheimer's risk in patients with metabolic disorders?

Authors:  Corey J Frank; Ewan C McNay
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3.  Expert Consensus on Cognitive Dysfunction in Diabetes.

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4.  Diabetes, Brain Infarcts, Cognition, and Small Vessels in the Canadian Alliance for Healthy Hearts and Minds Study.

Authors:  Hertzel C Gerstein; Eric E Smith; Chinthanie Ramasundarahettige; Dipika Desai; Philip Awadalla; Philippe Broet; Sandra Black; Trevor J B Dummer; Jason Hicks; Alan Moody; Jean-Claude Tardif; Koon K Teo; Jennifer Vena; Salim Yusuf; Douglas S Lee; Matthias G Friedrich; Sonia S Anand
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7.  Peganum harmala enhanced GLP-1 and restored insulin signaling to alleviate AlCl3-induced Alzheimer-like pathology model.

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Review 8.  Drug repositioning and repurposing for Alzheimer disease.

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Review 9.  GLP-1 receptor agonists (GLP-1RAs): cardiovascular actions and therapeutic potential.

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Journal:  Int J Biol Sci       Date:  2021-05-11       Impact factor: 6.580

10.  Systemic GLP-1R agonist treatment reverses mouse glial and neurovascular cell transcriptomic aging signatures in a genome-wide manner.

Authors:  Zhongqi Li; Xinyi Chen; Joaquim S L Vong; Lei Zhao; Junzhe Huang; Leo Y C Yan; Bonaventure Ip; Yun Kwok Wing; Hei-Ming Lai; Vincent C T Mok; Ho Ko
Journal:  Commun Biol       Date:  2021-06-02
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