Literature DB >> 32502225

Human exome and mouse embryonic expression data implicate ZFHX3, TRPS1, and CHD7 in human esophageal atresia.

Rong Zhang1, Jan Gehlen2, Amit Kawalia3, Maria-Theodora Melissari4, Tikam Chand Dakal5, Athira M Menon5, Julia Höfele6, Korbinian Riedhammer6,7, Lea Waffenschmidt1, Julia Fabian1, Katinka Breuer1, Jeshurun Kalanithy1, Alina Christine Hilger8, Amit Sharma9,10, Alice Hölscher11, Thomas M Boemers11, Markus Pauly12, Andreas Leutner13, Jörg Fuchs14, Guido Seitz15, Barbara M Ludwikowski16, Barbara Gomez16, Jochen Hubertus17, Andreas Heydweiller18, Ralf Kurz18, Johannes Leonhardt19, Ferdinand Kosch20, Stefan Holland-Cunz21, Oliver Münsterer22, Beno Ure23, Eberhard Schmiedeke24, Jörg Neser25, Petra Degenhardt26, Stefanie Märzheuser27, Katharina Kleine28, Mattias Schäfer29, Nicole Spychalski29, Oliver J Deffaa30, Jan-Hendrik Gosemann30, Martin Lacher30, Stefanie Heilmann-Heimbach1,31, Nadine Zwink32, Ekkehart Jenetzky32,33, Michael Ludwig34, Phillip Grote4, Johannes Schumacher1,2, Holger Thiele3, Heiko Reutter1,35.   

Abstract

INTRODUCTION: Esophageal atresia with or without tracheoesophageal fistula (EA/TEF) occurs approximately 1 in 3.500 live births representing the most common malformation of the upper digestive tract. Only half a century ago, EA/TEF was fatal among affected newborns suggesting that the steady birth prevalence might in parts be due to mutational de novo events in genes involved in foregut development.
METHODS: To identify mutational de novo events in EA/TEF patients, we surveyed the exome of 30 case-parent trios. Identified and confirmed de novo variants were prioritized using in silico prediction tools. To investigate the embryonic role of genes harboring prioritized de novo variants we performed targeted analysis of mouse transcriptome data of esophageal tissue obtained at the embryonic day (E) E8.5, E12.5, and postnatal.
RESULTS: In total we prioritized 14 novel de novo variants in 14 different genes (APOL2, EEF1D, CHD7, FANCB, GGT6, KIAA0556, NFX1, NPR2, PIGC, SLC5A2, TANC2, TRPS1, UBA3, and ZFHX3) and eight rare de novo variants in eight additional genes (CELSR1, CLP1, GPR133, HPS3, MTA3, PLEC, STAB1, and PPIP5K2). Through personal communication during the project, we identified an additional EA/TEF case-parent trio with a rare de novo variant in ZFHX3. In silico prediction analysis of the identified variants and comparative analysis of mouse transcriptome data of esophageal tissue obtained at E8.5, E12.5, and postnatal prioritized CHD7, TRPS1, and ZFHX3 as EA/TEF candidate genes. Re-sequencing of ZFHX3 in additional 192 EA/TEF patients did not identify further putative EA/TEF-associated variants.
CONCLUSION: Our study suggests that rare mutational de novo events in genes involved in foregut development contribute to the development of EA/TEF.

Entities:  

Year:  2020        PMID: 32502225     DOI: 10.1371/journal.pone.0234246

Source DB:  PubMed          Journal:  PLoS One        ISSN: 1932-6203            Impact factor:   3.240


  3 in total

Review 1.  Developmental basis of trachea-esophageal birth defects.

Authors:  Nicole A Edwards; Vered Shacham-Silverberg; Leelah Weitz; Paul S Kingma; Yufeng Shen; James M Wells; Wendy K Chung; Aaron M Zorn
Journal:  Dev Biol       Date:  2021-05-21       Impact factor: 3.582

2.  Diagnostic Value of Prenatal Ultrasound Parameters and Esophageal Signs in Pouch and Lower Thoracic Segment in Fetuses with Esophageal Atresia.

Authors:  Wenjun Feng
Journal:  Comput Math Methods Med       Date:  2021-12-22       Impact factor: 2.238

Review 3.  Heritability and De Novo Mutations in Oesophageal Atresia and Tracheoesophageal Fistula Aetiology.

Authors:  Erwin Brosens; Rutger W W Brouwer; Hannie Douben; Yolande van Bever; Alice S Brooks; Rene M H Wijnen; Wilfred F J van IJcken; Dick Tibboel; Robbert J Rottier; Annelies de Klein
Journal:  Genes (Basel)       Date:  2021-10-10       Impact factor: 4.096

  3 in total

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