Literature DB >> 32496962

Impact of tetraplegia vs. paraplegia on venoarteriolar, myogenic and maximal cutaneous vasodilation responses of the microvasculature: Implications for cardiovascular disease.

Michelle Trbovich1, Yubo Wu2, Wouter Koek3, Joan Zhao2, Dean Kellogg2.   

Abstract

Objective: Cardiovascular disease (CVD) is a leading cause of mortality in persons with SCI. While macrovascular remodeling and function after SCI is well documented, changes in the microvascular structure and function are comparably understudied, but importantly predict CVD risk. Specifically, the integrity of venoarteriolar (VAR), myogenic (MYO) and maximal vasodilation responses are largely unknown after SCI, especially in persons with tetraplegia (TP) at highest risk of CVD. This is the first to examine the differences in VAR (cuff inflation), MYO (limb dependency) and maximal vasodilation responses of the microvasculature between able bodied (AB) versus those with TP and paraplegia (PP).Design: Observational.Setting: Laboratory.Participants: Eight AB, 6 TP, and 8 PP persons.Interventions: One forearm and calf were treated topically with lidocaine 2.5%/prilocaine 2.5% while contralateral limb served as a control. Laser doppler flowmeters were applied over treated and control sites during limb dependency, cuff inflation and local skin heating (Tloc) up to 42°C.Outcome measures: Skin vascular resistance (SkVR) change with cuff inflation and limb dependency and maximal cutaneous vascular conductance (CVC) during local heating.
Results: Change in SkVR was not significantly different between groups or extremity (upper vs. lower) during cuff inflation or limb dependency. However, CVC at Tloc 42°C was significantly different in the lower extremity (LE) of TP and PP (P = 0.007, 0.35) compared to AB.
Conclusion: Increases in SkVR during cuff inflation (VAR) and limb dependency (VAR and MYO) are unaltered after SCI, however maximal vasodilation in the LE post-SCI is higher than AB persons.

Entities:  

Keywords:  Cardiovascular disease; Maximal cutaneous vasodilation; Micro-vasculature; Myogenic response; Orthostatic hypotension; Spinal cord injury; Sympathetic nervous system; Venoarteriolar response

Mesh:

Year:  2020        PMID: 32496962      PMCID: PMC8890560          DOI: 10.1080/10790268.2020.1761173

Source DB:  PubMed          Journal:  J Spinal Cord Med        ISSN: 1079-0268            Impact factor:   1.985


  56 in total

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Review 2.  Orthostatic hypotension following spinal cord injury: understanding clinical pathophysiology.

Authors:  V E Claydon; J D Steeves; A Krassioukov
Journal:  Spinal Cord       Date:  2005-11-22       Impact factor: 2.772

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Journal:  Acta Physiol Scand       Date:  1976-08

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Journal:  J Appl Physiol (1985)       Date:  2014-06-05

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Journal:  J Appl Physiol (1985)       Date:  2001-10

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Journal:  J Appl Physiol (1985)       Date:  1999-10

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Authors:  K Skagen; K Jensen; O Henriksen; L Knudsen
Journal:  Clin Sci (Lond)       Date:  1982-06       Impact factor: 6.124

8.  Leg intravenous pressure during head-up tilt.

Authors:  Jan T Groothuis; Fleur Poelkens; Constantijn W Wouters; Miriam Kooijman; Maria T E Hopman
Journal:  J Appl Physiol (1985)       Date:  2008-07-17

Review 9.  Peripheral vascular function in spinal cord injury: a systematic review.

Authors:  C R West; A Alyahya; I Laher; A Krassioukov
Journal:  Spinal Cord       Date:  2012-11-27       Impact factor: 2.772

10.  Effect of spinal sympathetic blockade upon postural changes of blood flow in human peripheral tissues.

Authors:  K Skagen; O Haxholdt; O Henriksen; V Dyrberg
Journal:  Acta Physiol Scand       Date:  1982-02
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