Literature DB >> 32487778

Androgen excess and diagnostic steroid biomarkers for nonclassic 21-hydroxylase deficiency without cosyntropin stimulation.

Adina F Turcu1, Diala El-Maouche2, Lili Zhao3, Aya T Nanba1, Alison Gaynor2, Padma Veeraraghavan2, Richard J Auchus1,4, Deborah P Merke2,5.   

Abstract

OBJECTIVES: The clinical presentation of patients with nonclassic 21-hydroxylase deficiency (N21OHD) is similar with that for other disorders of androgen excess. The diagnosis of N21OHD typically requires cosyntropin stimulation. Additionally, the management of such patients is limited by the lack of reliable biomarkers of androgen excess. Herein, we aimed to: (1.) compare the relative contribution of traditional and 11-oxyandrogens in N21OHD patients and (2.) identify steroids that accurately diagnose N21OHD with a single baseline blood draw.
DESIGN: We prospectively enrolled patients who underwent a cosyntropin stimulation test for suspected N21OHD in two tertiary referral centers between January 2016 and August 2019.
METHODS: Baseline sera were used to quantify 15 steroids by liquid chromatography-tandem mass spectrometry. Logistic regression modeling was implemented to select steroids that best discriminate N21OHD from controls.
RESULTS: Of 86 participants (72 females), median age 26, 32 patients (25 females) had N21OHD. Age, sex distribution, and BMI were similar between patients with N21OHD and controls. Both testosterone and androstenedione were similar in patients with N21OHD and controls, while four 11-oxyandrogens were significantly higher in patients with N21OHD (ratios between medians: 1.7 to 2.2, P < 0.01 for all). 17α-Hydroxyprogesterone (6.5-fold), 16α-hydroxyprogesterone (4.1-fold), and 21-deoxycortisol (undetectable in 80% of the controls) were higher, while corticosterone was 3.6-fold lower in patients with N21OHD than in controls (P < 0.001). Together, baseline 17α-hydroxyprogesterone, 21-deoxycortisol, and corticosterone showed perfect discrimination between N21OHD and controls.
CONCLUSIONS: Adrenal 11-oxyandrogens are disproportionately elevated compared to conventional androgens in N21OHD. Steroid panels can accurately diagnose N21OHD in unstimulated blood tests.

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Year:  2020        PMID: 32487778      PMCID: PMC7458124          DOI: 10.1530/EJE-20-0129

Source DB:  PubMed          Journal:  Eur J Endocrinol        ISSN: 0804-4643            Impact factor:   6.664


  38 in total

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2.  Profiles of 21-Carbon Steroids in 21-hydroxylase Deficiency.

Authors:  Adina F Turcu; Juilee Rege; Robert Chomic; Jiayan Liu; Hiromi K Nishimoto; Tobias Else; Andreas G Moraitis; Ganesh S Palapattu; William E Rainey; Richard J Auchus
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3.  Adrenal-derived 11-oxygenated 19-carbon steroids are the dominant androgens in classic 21-hydroxylase deficiency.

Authors:  Adina F Turcu; Aya T Nanba; Robert Chomic; Sunil K Upadhyay; Thomas J Giordano; James J Shields; Deborah P Merke; William E Rainey; Richard J Auchus
Journal:  Eur J Endocrinol       Date:  2016-02-10       Impact factor: 6.664

4.  Superior discriminating value of ACTH-stimulated serum 21-deoxycortisol in identifying heterozygote carriers for 21-hydroxylase deficiency.

Authors:  Flávia A Costa-Barbosa; Vânia F Tonetto-Fernandes; Valdemir M Carvalho; Odete H Nakamura; Vivian Moura; Tânia A S S Bachega; José G H Vieira; Claudio E Kater
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5.  Replication of clinical associations with 17-hydroxyprogesterone in preterm newborns.

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9.  Reassessing fecundity in women with classical congenital adrenal hyperplasia (CAH): normal pregnancy rate but reduced fertility rate.

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10.  Congenital Adrenal Hyperplasia Due to Steroid 21-Hydroxylase Deficiency: An Endocrine Society Clinical Practice Guideline.

Authors:  Phyllis W Speiser; Wiebke Arlt; Richard J Auchus; Laurence S Baskin; Gerard S Conway; Deborah P Merke; Heino F L Meyer-Bahlburg; Walter L Miller; M Hassan Murad; Sharon E Oberfield; Perrin C White
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2.  Maternal 11-Ketoandrostenedione Rises Through Normal Pregnancy and Is the Dominant 11-Oxygenated Androgen in Cord Blood.

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Review 4.  Nonclassic Congenital Adrenal Hyperplasia: What Do Endocrinologists Need to Know?

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5.  Analysis of a pitfall in congenital adrenal hyperplasia newborn screening: evidence of maternal use of corticoids detected on dried blood spot.

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6.  Production of 11-Oxygenated Androgens by Testicular Adrenal Rest Tumors.

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7.  24-Hour Profiles of 11-Oxygenated C19 Steroids and Δ5-Steroid Sulfates during Oral and Continuous Subcutaneous Glucocorticoids in 21-Hydroxylase Deficiency.

Authors:  Adina F Turcu; Ashwini Mallappa; Aikaterini A Nella; Xuan Chen; Lili Zhao; Aya T Nanba; James Brian Byrd; Richard J Auchus; Deborah P Merke
Journal:  Front Endocrinol (Lausanne)       Date:  2021-11-16       Impact factor: 5.555

8.  A Multiclassifier System to Identify and Subtype Congenital Adrenal Hyperplasia Based on Circulating Steroid Hormones.

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