| Literature DB >> 32437451 |
Daniel J Kostyniuk, Dapeng Zhang, Christopher J Martyniuk, Kathleen M Gilmour, Jan A Mennigen.
Abstract
[This corrects the article DOI: 10.1371/journal.pone.0217978.].Entities:
Year: 2020 PMID: 32437451 PMCID: PMC7241819 DOI: 10.1371/journal.pone.0233827
Source DB: PubMed Journal: PLoS One ISSN: 1932-6203 Impact factor: 3.240
Fig 7Steady state mRNA abundance (+S.E.M.) of genes involved in hepatic glucose metabolism, including gluconeogenic enzyme isoforms cytoplasmic phosphoenolpyruvate carboxykinase pck1 (A), mitochondrial phosphoenolpyruvate carboxykinase pck2 (B), gluconeogenic enzyme paralogues of glucose-6-phosphatase (C-G), and liver (H) and brain (I) isoforms of the glycogenolytic enzyme, glycogen phosphorylase. Data for SI, SD and SS rainbow trout (Oncorhynchus muykiss) were normalized using the Normagene algorithm, and then expressed relative to values for SI fish. A one-way ANOVAs followed by Tukey’s post-hoc was used for analysis. A p-value of p<0.05 was used as cut-off for significant effects.
Fig 5Relative steady-state abundance (+S.E.M.) of the most abundant differentially regulated hepatic miRNA, miRNA-21-5p, as well as miRNA-722, miRNA-26a-5p, miRNA-let-7a and miRNA-152 in liver of SD and SS rainbow trout, Oncorhynchus mykiss.
A one-tailed Welch’s t-test was used for analysis, and a p-value of p<0.05 was used as cut-off for significant effects.