Literature DB >> 32436162

Inhibition of neutrophil inflammatory mediator expression by azithromycin.

Monica P Gibson1,2, John D Walters3.   

Abstract

BACKGROUND AND
OBJECTIVE: Peri-implant tissues appear to exhibit a more vigorous inflammatory response during post-operative healing than periodontal tissues. There is evidence that a single dose of amoxicillin (AMX) prior to implant surgery reduces the risk of early peri-implant healing complications. This study compared the effects of AZM and AMX on neutrophil expression of mRNA for mediators involved in peri-implant healing.
MATERIALS AND METHODS: Neutrophils were isolated from healthy human donors and pre-incubated with AZM (4 or 8 μg/ml) or AMX (2 or 4 μg/ml). Cells were then incubated with LPS (1 μg/ml), TNF-α (10 ng/ml), or medium alone (control) for 1, 2, and 4 h. Total RNA was analyzed with qPCR to quantify changes in expression of the six inflammatory mediators.
RESULTS: LPS and TNF-α induced a similar pattern of IL-1β mRNA expression, with peak expression at 1 h. For most mediators, gene expression in neutrophils activated by LPS was markedly reduced in a dose-dependent manner by AZM. Therapeutic concentrations of AZM (8 μg/ml) consistently reduced expression of mediators tested in this study. AMX was effective only in a few cases and under certain conditions. Therefore, AZM was more effective in its direct anti-inflammatory action.
CONCLUSION: AZM is a consistent and effective inhibitor of neutrophil inflammatory mediator mRNA expression. CLINICAL RELEVANCE: Given that a single dose of AZM produces higher and more sustained concentrations of this agent in periodontal tissues than AMX when used as a pre-operative prophylactic antibiotic, AZM has greater potential to inhibit inflammatory mediator expression at peri-implant wound sites than AMX.

Entities:  

Keywords:  Antibiotic prophylaxis; Chemokines; Cytokines; Inflammation; Innate immunity

Mesh:

Substances:

Year:  2020        PMID: 32436162     DOI: 10.1007/s00784-020-03314-4

Source DB:  PubMed          Journal:  Clin Oral Investig        ISSN: 1432-6981            Impact factor:   3.573


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