Literature DB >> 32353608

Monitoring of asymptomatic family members at risk of hereditary transthyretin amyloidosis for early intervention with disease-modifying therapies.

Mitsuharu Ueda1, Yoshiki Sekijima2, Haruki Koike3, Taro Yamashita4, Tsuneaki Yoshinaga5, Tomonori Ishii6, Yukio Ando7.   

Abstract

BACKGROUND: Hereditary transthyretin (ATTRv) amyloidosis is an adult-onset, systemic disorder caused by mutations in the transthyretin (TTR) gene. As ATTRv amyloidosis is inherited in an autosomal dominant manner, family members of the patients are at risk of developing the disease.
METHODS: With an objective of discussing recommendations on monitoring of family members for early diagnosis of ATTRv amyloidosis, we held a medical advisory board meeting in Tokyo, Japan, in October 2017.
RESULTS: Our recommendations are summarized as follows: periodic follow-up genetic counseling should be offered to asymptomatic gene mutation carriers; follow-up assessments should be started when the carriers are still asymptomatic to test for amyloidosis onset, irrespective of TTR genotype and age at onset in the particular family. We suggest annual routine assessments and in-depth assessments every 3-5 years, with the frequency of these increased as required. Periodical monitoring of asymptomatic gene mutation carriers is crucial for attending physicians to detect early signs or symptoms of the disease and start disease-modifying therapy (DMT).
CONCLUSIONS: The monitoring strategy for asymptomatic TTR gene mutation carriers should progress toward rapid diagnosis and early intervention with DMT. This approach may be more appropriate for countries with more resources.
Copyright © 2020 The Authors. Published by Elsevier B.V. All rights reserved.

Entities:  

Keywords:  ATTRv amyloidosis; Asymptomatic; Early diagnosis; Genetic testing

Mesh:

Substances:

Year:  2020        PMID: 32353608     DOI: 10.1016/j.jns.2020.116813

Source DB:  PubMed          Journal:  J Neurol Sci        ISSN: 0022-510X            Impact factor:   3.181


  5 in total

1.  Three Newly Recognized Likely Pathogenic Gene Variants Associated with Hereditary Transthyretin Amyloidosis.

Authors:  Jignesh K Patel; Andrew M Rosen; Adam Chamberlin; Benjamin Feldmann; Christian Antolik; Heather Zimmermann; Tami Johnston; Arvind Narayana
Journal:  Neurol Ther       Date:  2022-08-06

2.  Plasma growth differentiation factor 15: a novel tool to detect early changes of hereditary transthyretin amyloidosis.

Authors:  Masamitsu Okada; Yohei Misumi; Teruaki Masuda; Seiji Takashio; Masayoshi Tasaki; Hiroaki Matsushita; Akihiko Ueda; Yasuteru Inoue; Toshiya Nomura; Makoto Nakajima; Taro Yamashita; Satoru Shinriki; Hirotaka Matsui; Kenichi Tsujita; Yukio Ando; Mitsuharu Ueda
Journal:  ESC Heart Fail       Date:  2020-12-30

Review 3.  Clinical recommendations to diagnose and monitor patients with transthyretin amyloid cardiomyopathy in Asia.

Authors:  Weiqin Lin; Pairoj Chattranukulchai; Alex Pw Lee; Yen-Hung Lin; Wen-Chung Yu; Houng-Bang Liew; Abraham Oomman
Journal:  Clin Cardiol       Date:  2022-07-06       Impact factor: 3.287

4.  Late-onset Hereditary ATTR Amyloidosis with a Novel p.P63S (P43S) Transthyretin Variant.

Authors:  Yuya Aono; Yasuhiro Hamatani; Nagaaki Katoh; Mayuko Nakagawa; Katsuya Nakamura; Masahide Yazaki; Fuyuki Kametani; Moritake Iguchi; Ikuko Murakami; Hisashi Ogawa; Mitsuru Abe; Masaharu Akao; Yoshiki Sekijima
Journal:  Intern Med       Date:  2020-09-30       Impact factor: 1.271

5.  Established and candidate transthyretin amyloidosis variants identified in the Saudi population by data mining.

Authors:  Mohamed Abouelhoda; Dania Mohty; Islam Alayary; Brian F Meyer; Stefan T Arold; Bahaa M Fadel; Dorota Monies
Journal:  Hum Genomics       Date:  2021-08-11       Impact factor: 4.639

  5 in total

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