Literature DB >> 32347164

ZO-1 Regulates Intercalated Disc Composition and Atrioventricular Node Conduction.

Le Shen1,2, Christopher R Weber1, Wenli Dai1, Rangarajan D Nadadur3, Jaclyn A Brennan4, Heather L Smith1, Kaitlyn M Shen3, Margaret Gadek3, Brigitte Laforest5, Mingyi Wang6, Joanna Gemel7, Ye Li1, Jing Zhang6, Bruce D Ziman6, Jiajie Yan8, Xun Ai8, Eric C Beyer7, Edward G Lakata6, Narayanan Kasthuri9, Igor R Efimov4, Michael T Broman5, Ivan P Moskowitz3.   

Abstract

RATIONALE: ZO-1 (Zona occludens 1), encoded by the tight junction protein 1 (TJP1) gene, is a regulator of paracellular permeability in epithelia and endothelia. ZO-1 interacts with the actin cytoskeleton, gap, and adherens junction proteins and localizes to intercalated discs in cardiomyocytes. However, the contribution of ZO-1 to cardiac physiology remains poorly defined.
OBJECTIVE: We aim to determine the role of ZO-1 in cardiac function. METHODS AND
RESULTS: Inducible cardiomyocyte-specific Tjp1 deletion mice (Tjp1fl/fl; Myh6Cre/Esr1*) were generated by crossing the Tjp1 floxed mice and Myh6Cre/Esr1* transgenic mice. Tamoxifen-induced loss of ZO-1 led to atrioventricular (AV) block without changes in heart rate, as measured by ECG and ex vivo optical mapping. Mice with tamoxifen-induced conduction system-specific deletion of Tjp1 (Tjp1fl/fl; Hcn4CreERt2) developed AV block while tamoxifen-induced conduction system deletion of Tjp1 distal to the AV node (Tjp1fl/fl; Kcne1CreERt2) did not demonstrate conduction defects. Western blot and immunostaining analyses of AV nodes showed that ZO-1 loss decreased Cx (connexin) 40 expression and intercalated disc localization. Consistent with the mouse model study, immunohistochemical staining showed that ZO-1 is abundantly expressed in the human AV node and colocalizes with Cx40. Ventricular conduction was not altered despite decreased localization of ZO-1 and Cx43 at the ventricular intercalated disc and modestly decreased left ventricular ejection fraction, suggesting ZO-1 is differentially required for AV node and ventricular conduction.
CONCLUSIONS: ZO-1 is a key protein responsible for maintaining appropriate AV node conduction through maintaining gap junction protein localization.

Entities:  

Keywords:  atrioventricular block; connexins; cytoskeleton; intercellular junctions; tight junctions

Mesh:

Substances:

Year:  2020        PMID: 32347164      PMCID: PMC7334106          DOI: 10.1161/CIRCRESAHA.119.316415

Source DB:  PubMed          Journal:  Circ Res        ISSN: 0009-7330            Impact factor:   17.367


  68 in total

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10.  Gap junction remodelling in human heart failure is associated with increased interaction of connexin43 with ZO-1.

Authors:  Alexandra F Bruce; Stephen Rothery; Emmanuel Dupont; Nicholas J Severs
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