Literature DB >> 32241770

Brain Metastases: Insights from Statistical Modeling of Size Distribution.

M Buller1, K M Chapple1, C R Bird2.   

Abstract

BACKGROUND AND
PURPOSE: Brain metastases are a common finding on brain MRI. However, the factors that dictate their size and distribution are incompletely understood. Our aim was to discover a statistical model that can account for the size distribution of parenchymal metastases in the brain as measured on contrast-enhanced MR imaging.
MATERIALS AND METHODS: Tumor volumes were calculated on the basis of measured tumor diameters from contrast-enhanced T1-weighted spoiled gradient-echo images in 68 patients with untreated parenchymal metastatic disease. Tumor volumes were then placed in rank-order distributions and compared with 11 different statistical curve types. The resultant R 2 values to assess goodness of fit were calculated. The top 2 distributions were then compared using the likelihood ratio test, with resultant R values demonstrating the relative likelihood of these distributions accounting for the observed data.
RESULTS: Thirty-nine of 68 cases best fit a power distribution (mean R 2 = 0.938 ± 0.050), 20 cases best fit an exponential distribution (mean R 2 = 0.957 ± 0.050), and the remaining cases were scattered among the remaining distributions. Likelihood ratio analysis revealed that 66 of 68 cases had a positive mean R value (1.596 ± 1.316), skewing toward a power law distribution.
CONCLUSIONS: The size distributions of untreated brain metastases favor a power law distribution. This finding suggests that metastases do not exist in isolation, but rather as part of a complex system. Furthermore, these results suggest that there may be a relatively small number of underlying variables that substantially influence the behavior of these systems. The identification of these variables could have a profound effect on our understanding of these lesions and our ability to treat them.
© 2020 by American Journal of Neuroradiology.

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Mesh:

Year:  2020        PMID: 32241770      PMCID: PMC7144648          DOI: 10.3174/ajnr.A6496

Source DB:  PubMed          Journal:  AJNR Am J Neuroradiol        ISSN: 0195-6108            Impact factor:   3.825


  13 in total

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2.  Common ecology quantifies human insurgency.

Authors:  Juan Camilo Bohorquez; Sean Gourley; Alexander R Dixon; Michael Spagat; Neil F Johnson
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5.  Tyrosine isomers mediate the classical phenomenon of concomitant tumor resistance.

Authors:  Raúl A Ruggiero; Juan Bruzzo; Paula Chiarella; Pedro di Gianni; Martín A Isturiz; Susana Linskens; Norma Speziale; Roberto P Meiss; Oscar D Bustuoabad; Christiane D Pasqualini
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7.  Mathematical Modeling of Tumor-Tumor Distant Interactions Supports a Systemic Control of Tumor Growth.

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Review 8.  Information dynamics in carcinogenesis and tumor growth.

Authors:  Robert A Gatenby; B Roy Frieden
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9.  Angiostatin: a novel angiogenesis inhibitor that mediates the suppression of metastases by a Lewis lung carcinoma.

Authors:  M S O'Reilly; L Holmgren; Y Shing; C Chen; R A Rosenthal; M Moses; W S Lane; Y Cao; E H Sage; J Folkman
Journal:  Cell       Date:  1994-10-21       Impact factor: 41.582

10.  The growth law of primary breast cancer as inferred from mammography screening trials data.

Authors:  D Hart; E Shochat; Z Agur
Journal:  Br J Cancer       Date:  1998-08       Impact factor: 7.640

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