Literature DB >> 32215465

Involvement of HSP90 in ischemic postconditioning-induced cardioprotection by inhibition of the complement system, JNK and inflammation.

Dong-Xiao Wang1, Zheng Huang2, Qing-Jie Li3, Guo-Qiang Zhong4, Yan He5, Wei-Qiang Huang6, Xiao-Li Cao7, Rong-Hui Tu8, Jian-Jun Meng9.   

Abstract

PURPOSE: To investigate whether heat shock protein 90 (HSP90) is involved in complement regulation in ischemic postconditioning (IPC).
METHODS: The left coronary artery of rats underwent 30 min of occlusion, followed by 120 min of reperfusion and treatment with IPC via 3 cycles of 30s reperfusion and 30s occlusion. The rats were injected intraperitoneally with 1 mg/kg HSP90 inhibitor geldanamycin (GA) after anesthesia. Eighty rats were randomly divided into four groups: sham, ischemia-reperfusion (I/R), IPC and IPC + GA. Myocardial infarct size, apoptosis index and the expression of HSP90, C3, C5a, tumor necrosis factor (TNF)-alpha, interleukin (IL)-1β and c-Jun N-terminal kinase (JNK) were assessed.
RESULTS: Compared with the I/R injury, the IPC treatment significantly reduced infarct size, release of troponin T, creatine kinase-MB, and lactate dehydrogenase, and cardiomyocyte apoptosis. These beneficial effects were accompanied by a decrease in TNF-α, IL-1β, C3, C5a and JNK expression levels. However, all these effects were abrogated by administration of the HSP90 inhibitor GA.
CONCLUSION: HSP90 exerts a profound effect on IPC cardioprotection, and may be linked to the inhibition of the complement system and JNK, ultimately attenuating I/R-induced myocardial injury and apoptosis.

Entities:  

Year:  2020        PMID: 32215465     DOI: 10.1590/s0102-865020200010000005

Source DB:  PubMed          Journal:  Acta Cir Bras        ISSN: 0102-8650            Impact factor:   1.388


  6 in total

1.  Klotho overexpression suppresses apoptosis by regulating the Hsp70/Akt/Bad pathway in H9c2(2-1) cells.

Authors:  Jinpeng Hu; Bin Su; Xuewen Li; Yuming Li; Jihong Zhao
Journal:  Exp Ther Med       Date:  2021-03-16       Impact factor: 2.447

2.  HSF1 is involved in suppressing A1 phenotype conversion of astrocytes following spinal cord injury in rats.

Authors:  Lilan Li; Yu Li; Bingqiang He; Hui Li; Huiyuan Ji; Yingjie Wang; Zhenjie Zhu; Yuming Hu; Yue Zhou; Ting Yang; Chunshuai Sun; Ying Yuan; Yongjun Wang
Journal:  J Neuroinflammation       Date:  2021-09-16       Impact factor: 8.322

3.  The FcεRI signaling pathway is involved in the pathogenesis of lacrimal gland benign lymphoepithelial lesions as shown by transcriptomic analysis.

Authors:  Jing Li; Rui Liu; Mei Sun; Jinjin Wang; Nan Wang; Xuan Zhang; Xin Ge; Jianmin Ma
Journal:  Sci Rep       Date:  2021-11-08       Impact factor: 4.379

4.  New targets of morphine postconditioning protection of the myocardium in ischemia/reperfusion injury: Involvement of HSP90/Akt and C5a/NF-κB.

Authors:  Rong-Hui Tu; Dong-Xiao Wang; Guo-Qiang Zhong; Jian-Jun Meng; Hong Wen; Qi Bi; Yan He
Journal:  Open Med (Wars)       Date:  2021-10-18

Review 5.  Heat Shock Proteins: Potential Modulators and Candidate Biomarkers of Peripartum Cardiomyopathy.

Authors:  Graham Chakafana; Timothy F Spracklen; Stephen Kamuli; Tawanda Zininga; Addmore Shonhai; Ntobeko A B Ntusi; Karen Sliwa
Journal:  Front Cardiovasc Med       Date:  2021-06-16

Review 6.  Heat Shock Protein 90 as Therapeutic Target for CVDs and Heart Ageing.

Authors:  Siarhei A Dabravolski; Vasily N Sukhorukov; Vladislav A Kalmykov; Nikolay A Orekhov; Andrey V Grechko; Alexander N Orekhov
Journal:  Int J Mol Sci       Date:  2022-01-07       Impact factor: 5.923

  6 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.