Literature DB >> 32196695

Reduced Hippocampal Volumes Partially Mediate Effects of Prenatal Alcohol Exposure on Spatial Navigation on a Virtual Water Maze Task in Children.

Neil C Dodge1, Kevin G F Thomas2, Ernesta M Meintjes3, Christopher D Molteno4, Joseph L Jacobson1,3,4, Sandra W Jacobson1,3,4.   

Abstract

BACKGROUND: Prenatal alcohol exposure (PAE) has been linked to poorer performance on the Morris water maze (MWM), a test of spatial navigation in rodents that is dependent on hippocampal functioning. We recently confirmed these findings in children with PAE on a human analog of the MWM, the virtual water maze (VWM). Previous studies have shown that the hippocampus is particularly sensitive to PAE. Our aim was to determine whether hippocampal volume mediates the relation between PAE and virtual navigation.
METHODS: VWM and MRI hippocampal data were collected from 50 right-handed 10-year-old children in a heavily exposed Cape Town, South African sample. PAE data had been collected from their mothers during pregnancy, and the children were examined by expert fetal alcohol spectrum disorder (FASD) dysmorphologists. In the VWM, the participant attempts to learn the location of a hidden platform in a virtual pool of water across a series of learning trials using only distal room cues. Hippocampal volumes were derived using FreeSurfer from MRI scans administered within 1 week of completing the VWM task.
RESULTS: Both the fetal alcohol syndrome (FAS)/partial FAS and nonsyndromal heavy-exposed (HE) groups had smaller hippocampal volumes than controls. PAE was associated with reduced right hippocampal volumes even after control for total intracranial volume (ICV). Hippocampal volume was also positively associated with VWM performance. The relation between PAE and VWM performance was partially mediated by right hippocampal volume but not by total ICV.
CONCLUSIONS: These data confirm previous reports linking PAE to poorer spatial navigation on the VWM and are the first to provide direct evidence that volume reductions in this region partially mediate the relation of FASD diagnosis to place learning, suggesting that PAE specifically impairs the ability to encode the spatial information necessary for successful location of the hidden platform on a navigation task.
© 2020 by the Research Society on Alcoholism.

Entities:  

Keywords:  Fetal Alcohol Spectrum Disorders; Fetal Alcohol Syndrome; Hippocampal Volume; Place Learning; Prenatal Alcohol Exposure; Spatial Navigation; Virtual Environment

Mesh:

Year:  2020        PMID: 32196695      PMCID: PMC7166198          DOI: 10.1111/acer.14310

Source DB:  PubMed          Journal:  Alcohol Clin Exp Res        ISSN: 0145-6008            Impact factor:   3.455


  56 in total

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Authors:  Sandra W Jacobson; Mark E Stanton; Neil C Dodge; Mariska Pienaar; Douglas S Fuller; Christopher D Molteno; Ernesta M Meintjes; H Eugene Hoyme; Luther K Robinson; Nathaniel Khaole; Joseph L Jacobson
Journal:  Alcohol Clin Exp Res       Date:  2010-11-12       Impact factor: 3.455

2.  Three-dimensional surface deformation-based shape analysis of hippocampus and caudate nucleus in children with fetal alcohol spectrum disorders.

Authors:  Jesuchristopher Joseph; Christopher Warton; Sandra W Jacobson; Joseph L Jacobson; Chris D Molteno; Anton Eicher; Patrick Marais; Owen R Phillips; Katherine L Narr; Ernesta M Meintjes
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Authors:  Sandra W Jacobson; Lisa M Chiodo; Robert J Sokol; Joseph L Jacobson
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4.  Lateralized human hippocampal activity predicts navigation based on sequence or place memory.

Authors:  Kinga Iglói; Christian F Doeller; Alain Berthoz; Laure Rondi-Reig; Neil Burgess
Journal:  Proc Natl Acad Sci U S A       Date:  2010-07-26       Impact factor: 11.205

5.  Deficits on a spatial navigation task following prenatal exposure to ethanol.

Authors:  B A Blanchard; E P Riley; J H Hannigan
Journal:  Neurotoxicol Teratol       Date:  1987 May-Jun       Impact factor: 3.763

6.  Spatial memory deficits in patients with lesions to the right hippocampus and to the right parahippocampal cortex.

Authors:  V D Bohbot; M Kalina; K Stepankova; N Spackova; M Petrides; L Nadel
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7.  Magnetic resonance imaging outcomes from a comprehensive magnetic resonance study of children with fetal alcohol spectrum disorders.

Authors:  Susan J Astley; Elizabeth H Aylward; Heather Carmichael Olson; Kimberly Kerns; Allison Brooks; Truman E Coggins; Julian Davies; Susan Dorn; Beth Gendler; Tracy Jirikowic; Paul Kraegel; Kenneth Maravilla; Todd Richards
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8.  Validation of a new biomarker of fetal exposure to alcohol.

Authors:  Cynthia F Bearer; Joseph L Jacobson; Sandra W Jacobson; Dana Barr; Julie Croxford; Christopher D Molteno; Denis L Viljoen; Anna-Susan Marais; Lisa M Chiodo; Andrew S Cwik
Journal:  J Pediatr       Date:  2003-10       Impact factor: 4.406

9.  Developments of a water-maze procedure for studying spatial learning in the rat.

Authors:  R Morris
Journal:  J Neurosci Methods       Date:  1984-05       Impact factor: 2.390

10.  A study of cortical morphology in children with fetal alcohol spectrum disorders.

Authors:  François De Guio; Jean-François Mangin; Denis Rivière; Matthieu Perrot; Christopher D Molteno; Sandra W Jacobson; Ernesta M Meintjes; Joseph L Jacobson
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2.  Hippocampal subfield abnormalities and memory functioning in children with fetal alcohol Spectrum disorders.

Authors:  Donovan J Roediger; Alyssa M Krueger; Erik de Water; Bryon A Mueller; Christopher A Boys; Timothy J Hendrickson; Mariah J Schumacher; Sarah N Mattson; Kenneth L Jones; Kelvin O Lim; Jeffrey R Wozniak
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3.  Functional and Structural Correlates of Impaired Enrichment-Mediated Adult Hippocampal Neurogenesis in a Mouse Model of Prenatal Alcohol Exposure.

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4.  Ethanol modulation of hippocampal neuroinflammation, myelination, and neurodevelopment in a postnatal mouse model of fetal alcohol spectrum disorders.

Authors:  Victoria M Niedzwiedz-Massey; James C Douglas; Tonya Rafferty; Patricia A Wight; Cynthia J M Kane; Paul D Drew
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  4 in total

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