Literature DB >> 32192998

Computer modeling of pulsatile blood flow in elastic artery using a software program for application in biomedical engineering.

Bahador Sharifzadeh1, Rasool Kalbasi1, Mehdi Jahangiri2, Davood Toghraie3, Arash Karimipour4.   

Abstract

BACKGROUND AND
OBJECTIVE: Atherosclerosis-a condition in which an artery is constricted-alters blood flow in the artery, that can exacerbate the condition. Focusing on previous studies, it can be seen that the k-ε model has been used in the simulation. Therefore, the reverse flow on the back of stenosis is not well represented. In this study, the simulated results are much closer to clinical results, relying on the use of physiological pulses, and considering elasticity of the vessel wall, and the applying k-ω model. It can therefore be claimed that a much more accurate prediction will be made regarding the formation, development and progression of the disease.
METHODS: Modeling biological systems usually contain many parameters, which cannot be calculated experimentally, or are too costly and time consuming. In addition, it is occasionally required to examine the influence of different physical variables, which, given the complexity of the governing equations, make analytical methods feasible (or very limited). The present study is an attempt to investigate the turbulent pulsatile blood flow in an elastic artery with single and double stenoses using a finite element software program, ADINA 8.8.
RESULTS: According to the results, the k - ω turbulence model predicted a larger reverse flow in the post-stenotic region and between the two stenoses in comparison with the k - ε model. In other words, the k - ω model results suggest that a larger region is prone to atherosclerosis. In addition, that the k - ε model predicted a greater maximum shear stress at the throat and a shorter reverse flow region (Mean WSS < 0) in both stenosis scenarios. In other words, relative to the k - ε model, the k - ω model underestimated the damage to the plaque and the risk of its rupture though it predicted new stenosis developing behind the previous one. It was observed that the presence of a double stenosis causes the upstream pressure to reach the critical value in less time. Velocity profiles revealed that in the stenosis throat, the maximum velocity exceeds the normal biological state, which may cause disorders in the blood circulation.
CONCLUSIONS: The artery wall displacement results are suggestive of the greater difference between the two turbulence models in the case with double stenosis compared with single stenosis. Moreover, the difference between the two turbulence models in double stenosis is minimized in both post-stenotic and pre-stenotic regions.
Copyright © 2020 Elsevier B.V. All rights reserved.

Entities:  

Keywords:  Axial pressure drop; Blood-Artery interactions; Double stenosis; Shear stress; Single stenosis

Mesh:

Year:  2020        PMID: 32192998     DOI: 10.1016/j.cmpb.2020.105442

Source DB:  PubMed          Journal:  Comput Methods Programs Biomed        ISSN: 0169-2607            Impact factor:   5.428


  2 in total

1.  Effect of Extended Lipid Core on the Hemodynamic Parameters: A Fluid-Structure Interaction Approach.

Authors:  Morteza Teymoori; Mahmood Reza Sadeghi; Mohsen Rabbani; Mehdi Jahangiri
Journal:  Appl Bionics Biomech       Date:  2022-03-17       Impact factor: 1.781

2.  Investigation of Artery Wall Elasticity Effect on the Prediction of Atherosclerosis by Hemodynamic Factors.

Authors:  Rasool Kalbasi; Bahador Sharifzadeh; Mehdi Jahangiri
Journal:  Appl Bionics Biomech       Date:  2022-04-05       Impact factor: 1.781

  2 in total

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