Literature DB >> 3215746

Ataxic hemiparesis syndrome: sensory disturbances and somatosensory evoked potentials.

V Crespi1, G Bogliun, M L Delodovici, I Sanguineti.   

Abstract

26 patients with ataxic hemiparesis syndrome (AHS), due to acute ischemic cerebrovascular disease, have been submitted to clinical and electrophysiological evaluation, in order to assess the frequency of sensory disturbances in this condition. Sensory impairment were present in 78% and SEP abnormalities in 54% of the patients, while they were entirely absent in 23% of them. Lesions responsible for AHS, detected by CT scan, were mainly located in the thalamus, capsula interna, subcortical white matter, centro parietal cortex; sensory and SEP changes were more frequent in gross infarct involving the cortex and in smaller infarcts involving the thalamus, less frequent in the lacunar infarcts of the capsula interna and subcortical white matter, relatively rare in patients with CT scan without hypodense lesions. Although a statokinesthesic defect and/or major SEP abnormalities were often present (38% of patients), our findings do not support the view that they are involved in the pathogenesis of the ataxia, which may rather be attributed to a derangement of cerebro-cerebellar and cerebello-cerebral connections.

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Year:  1988        PMID: 3215746     DOI: 10.1007/bf02337163

Source DB:  PubMed          Journal:  Ital J Neurol Sci        ISSN: 0392-0461


  22 in total

1.  Somatosensory cerebral evoked potentials after vascular lesions of the brain-stem and diencephalon.

Authors:  P Noël; J E Desmedt
Journal:  Brain       Date:  1975-03       Impact factor: 13.501

2.  The significance of somatosensory evoked potentials for localization of unilateral lesions within the cerebral hemispheres.

Authors:  M Stöhr; J Dichgans; K Voigt; U W Buettner
Journal:  J Neurol Sci       Date:  1983-09       Impact factor: 3.181

3.  Non-cephalic reference recording of early somatosensory potentials to finger stimulation in adult or aging normal man: differentiation of widespread N18 and contralateral N20 from the prerolandic P22 and N30 components.

Authors:  J E Desmedt; G Cheron
Journal:  Electroencephalogr Clin Neurophysiol       Date:  1981-12

4.  Ataxic hemiparesis: clinical, electrophysiologic, radiologic and pathologic observations.

Authors:  K Radhakrishnan; A K Malhotra; R Shridharan; J S Chopra; A K Banerjee
Journal:  Clin Neurol Neurosurg       Date:  1982       Impact factor: 1.876

5.  Capsular ataxic hemiparesis. A case report.

Authors:  K Ichikawa; A Tsutsumishita; A Fujioka
Journal:  Arch Neurol       Date:  1982-09

6.  Short-latency somatosensory-evoked potentials in patients with acute focal vascular lesions of the supratentorial somesthesic pathways.

Authors:  V Crespi; A Mandelli; G Minoli
Journal:  Acta Neurol Scand       Date:  1982-04       Impact factor: 3.209

7.  A lacunar stroke. The dysarthria-clumsy hand syndrome.

Authors:  C M Fisher
Journal:  Neurology       Date:  1967-06       Impact factor: 9.910

8.  Homolateral ataxia and crural paresis: case report.

Authors:  G P Perman; A Racy
Journal:  Neurology       Date:  1980-09       Impact factor: 9.910

9.  A prospective study of lacunar infarction using computerized tomography.

Authors:  G A Donnan; B M Tress; P F Bladin
Journal:  Neurology       Date:  1982-01       Impact factor: 9.910

10.  Gating of somatosensory evoked potentials during different kinds of movement in man.

Authors:  D N Rushton; J C Rothwell; M D Craggs
Journal:  Brain       Date:  1981-09       Impact factor: 13.501

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