Literature DB >> 32098856

Defining research priorities in dystonia.

Codrin Lungu1, Laurie Ozelius2, David Standaert2, Mark Hallett2, Beth-Anne Sieber2, Christine Swanson-Fisher2, Brian D Berman2, Nicole Calakos2, Jennifer C Moore2, Joel S Perlmutter2, Sarah E Pirio Richardson2, Rachel Saunders-Pullman2, Laura Scheinfeldt2, Nutan Sharma2, Roy Sillitoe2, Kristina Simonyan2, Philip A Starr2, Anna Taylor2, Jerrold Vitek2.   

Abstract

OBJECTIVE: Dystonia is a complex movement disorder. Research progress has been difficult, particularly in developing widely effective therapies. This is a review of the current state of knowledge, research gaps, and proposed research priorities.
METHODS: The NIH convened leaders in the field for a 2-day workshop. The participants addressed the natural history of the disease, the underlying etiology, the pathophysiology, relevant research technologies, research resources, and therapeutic approaches and attempted to prioritize dystonia research recommendations.
RESULTS: The heterogeneity of dystonia poses challenges to research and therapy development. Much can be learned from specific genetic subtypes, and the disorder can be conceptualized along clinical, etiology, and pathophysiology axes. Advances in research technology and pooled resources can accelerate progress. Although etiologically based therapies would be optimal, a focus on circuit abnormalities can provide a convergent common target for symptomatic therapies across dystonia subtypes. The discussions have been integrated into a comprehensive review of all aspects of dystonia.
CONCLUSION: Overall research priorities include the generation and integration of high-quality phenotypic and genotypic data, reproducing key features in cellular and animal models, both of basic cellular mechanisms and phenotypes, leveraging new research technologies, and targeting circuit-level dysfunction with therapeutic interventions. Collaboration is necessary both for collection of large data sets and integration of different research methods.
© 2020 American Academy of Neurology.

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Year:  2020        PMID: 32098856      PMCID: PMC7274927          DOI: 10.1212/WNL.0000000000009140

Source DB:  PubMed          Journal:  Neurology        ISSN: 0028-3878            Impact factor:   9.910


  59 in total

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2.  The early-onset torsion dystonia gene (DYT1) encodes an ATP-binding protein.

Authors:  L J Ozelius; J W Hewett; C E Page; S B Bressman; P L Kramer; C Shalish; D de Leon; M F Brin; D Raymond; D P Corey; S Fahn; N J Risch; A J Buckler; J F Gusella; X O Breakefield
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Authors:  Cathérine C S Delnooz; Jaco W Pasman; Christian F Beckmann; Bart P C van de Warrenburg
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  7 in total

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