Literature DB >> 32094078

Clinical, radiological, and pathological features of anti-asparaginyl tRNA synthetase antibody-related interstitial lung disease.

Naoto Aiko1, Hideaki Yamakawa2, Tae Iwasawa3, Tamiko Takemura4, Koji Okudela5, Hideya Kitamura6, Eri Hagiwara6, Satoshi Ikeda6, Tomohisa Baba6, Shinichiro Iso7, Yukie Yamaguchi8, Yasushi Kondo9, Takayoshi Kurabayashi9, Kenichi Ohashi5, Shinji Sato9, Takashi Ogura6.   

Abstract

BACKGROUND: Myositis and interstitial lung disease (ILD) frequently occur in patients with anti-aminoacyl-tRNA synthetase (ARS) antibodies. Nearly half of ARS-ILD patients have the acute or subacute form of the disease, and one-third of these patients show a deterioration in pulmonary function over the long-term course because of frequent recurrences and refractoriness to therapy. Several reports recently described different characteristics depending on the individual anti-ARS antibodies, and the anti-asparaginyl tRNA synthetase (KS) antibody was strongly linked to ILD rather than to myositis. We therefore hypothesized that KS-ILD may have clinical characteristics that differ from those of other ARS-ILDs. The aim of this study was to clarify the clinical, radiological, and pathological features of KS antibody-positive ILD.
METHODS: We retrospectively analyzed 19 consecutive patients with KS-ILD who underwent initial clinical measurements and high-resolution computed tomography and pathological assessments. We also analyzed disease behavior based on pulmonary function test results during the follow-up period.
RESULTS: Our KS-ILD cohort included patients with dermatomyositis (10.5%), primary Sjögren syndrome (5.3%), and idiopathic ILD (84.2%). Most patients presented with chronic onset (89.5%) and a nonspecific pattern of interstitial pneumonia at each radiological and pathological assessment (89.4% and 85.7%, respectively). The pulmonary function test results showed that the mean changes from the initial %forced vital capacity and %diffusing capacity of the lung for carbon monoxide at 3 years were 3.7% ± 2.9% and 9.35% ± 3.0%, respectively.
CONCLUSIONS: Most KS-ILD patients showed a tendency for chronic disease onset and long-term stabilization of pulmonary function.
Copyright © 2020 The Japanese Respiratory Society. Published by Elsevier B.V. All rights reserved.

Entities:  

Keywords:  Anti-asparaginyl tRNA synthetase (anti-KS) antibody; Anti-synthetase syndrome; Dermatomyositis; Idiopathic interstitial pneumonia

Year:  2020        PMID: 32094078     DOI: 10.1016/j.resinv.2019.12.003

Source DB:  PubMed          Journal:  Respir Investig        ISSN: 2212-5345


  4 in total

1.  Interstitial lung disease is not rare in immune-mediated necrotizing myopathy with anti-signal recognition particle antibodies.

Authors:  Yongpeng Ge; Hanbo Yang; Xinyue Xiao; Lin Liang; Xin Lu; Guochun Wang
Journal:  BMC Pulm Med       Date:  2022-01-10       Impact factor: 3.317

Review 2.  Interstitial lung disease is a major characteristic of anti-KS associated ant-synthetase syndrome.

Authors:  Yongpeng Ge; Sizhao Li; Shanshan Li; Linrong He; Xin Lu; Guochun Wang
Journal:  Ther Adv Chronic Dis       Date:  2020-10-29       Impact factor: 5.091

3.  Prevalence of Novel Myositis Autoantibodies in a Large Cohort of Patients with Interstitial Lung Disease.

Authors:  Sofia A Moll; Mark G J P Platenburg; Anouk C M Platteel; Adriane D M Vorselaars; Montse Janssen Bonàs; Claudia Roodenburg-Benschop; Bob Meek; Coline H M van Moorsel; Jan C Grutters
Journal:  J Clin Med       Date:  2020-09-11       Impact factor: 4.241

4.  Interstitial Lung Disease Is a Major Characteristic of Patients Who Test Positive for Anti-PM/Scl Antibody.

Authors:  Yongpeng Ge; Xiaoming Shu; Linrong He; Chunjia Li; Xin Lu; Guochun Wang
Journal:  Front Med (Lausanne)       Date:  2022-01-18
  4 in total

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