Literature DB >> 32068183

Mechanisms of atrial fibrillation in aged rats with heart failure with preserved ejection fraction.

Thassio Ricardo Ribeiro Mesquita1, Rui Zhang2, Geoffrey de Couto1, Jackelyn Valle1, Lizbeth Sanchez1, Russell G Rogers1, Kevin Holm1, Weixin Liu1, Eduardo Marbán1, Eugenio Cingolani3.   

Abstract

BACKGROUND: Although ∼20% of the elderly population develops atrial fibrillation (AF), little is known about the mechanisms. Heart failure with preserved ejection fraction (HFpEF), which is associated with AF, is more common in aged women than in men.
OBJECTIVE: The purpose of this study was to identify potential mechanisms of AF in an age-related HFpEF model.
METHODS: In aged female Fischer F344 rats (21- to 24-month-old), which are prone to HFpEF, we induced AF by atrial pacing. Young Fischer F344 female rats (3- to 4-month-old) and age-matched Sprague Dawley female rats (27-month-old) served as controls. Phenotyping included echocardiography to assess left ventricular structure/function; in vivo electrophysiology and ex vivo high-resolution optical mapping to assess AF vulnerability; systemic and atrial inflammatory profiling; atrial histology; and expression of inflammasome signaling proteins.
RESULTS: Aged rats developed left ventricular hypertrophy, left atrial enlargement, diastolic dysfunction, and pulmonary congestion, without ejection fraction impairment, thus meeting the criteria for HFpEF. Increased serum inflammatory markers, hypertension, and obesity further characterize aged females. Sinoatrial and atrioventricular node dysfunction was associated with the high inducibility of AF in aged rats. Ex vivo electrical activation mapping revealed abnormal β-adrenergic responsiveness and slowed conduction velocity. Atrial inflammasome signaling was enhanced in aged rats, which may contribute to fibrotic remodeling and high AF susceptibility.
CONCLUSION: Together, our data demonstrate that aging-related atrial remodeling and HFpEF are associated with atrial enlargement, fibrosis, conduction abnormalities, and nodal dysfunction, favoring a substrate conducive to AF.
Copyright © 2020 Heart Rhythm Society. Published by Elsevier Inc. All rights reserved.

Entities:  

Keywords:  Aging; Atrial fibrillation; Fibrosis; HFpEF; Inflammation

Mesh:

Year:  2020        PMID: 32068183      PMCID: PMC7802887          DOI: 10.1016/j.hrthm.2020.02.007

Source DB:  PubMed          Journal:  Heart Rhythm        ISSN: 1547-5271            Impact factor:   6.343


  31 in total

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Journal:  Circ Heart Fail       Date:  2010-11-12       Impact factor: 8.790

Review 2.  Epidemiology of heart failure with preserved ejection fraction.

Authors:  Shannon M Dunlay; Véronique L Roger; Margaret M Redfield
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3.  Ventricular Arrhythmias Underlie Sudden Death in Rats With Heart Failure and Preserved Ejection Fraction.

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Authors:  Frederick A Masoudi; Edward P Havranek; Grace Smith; Ronald H Fish; John F Steiner; Diana L Ordin; Harlan M Krumholz
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Journal:  J Exp Med       Date:  2018-01-16       Impact factor: 14.307

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2.  Mechanisms of Sinoatrial Node Dysfunction in Heart Failure With Preserved Ejection Fraction.

Authors:  Thassio Mesquita; Rui Zhang; Jae Hyung Cho; Rui Zhang; Yen-Nien Lin; Lizbeth Sanchez; Joshua I Goldhaber; Joseph K Yu; Jialiu A Liang; Weixin Liu; Natalia A Trayanova; Eugenio Cingolani
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Review 8.  Chronic low-grade inflammation in heart failure with preserved ejection fraction.

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  8 in total

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