Corrigendum to "TERT and Akt Are Involved in the Par-4-Dependent Apoptosis of Islet β Cells in Type 2 Diabetes".

[This corrects the article DOI: 10.1155/2018/7653904.].

In the article titled “TERT and Akt Are Involved in the Par-4-Dependent Apoptosis of Islet β Cells in Type 2 Diabetes” [1], the image of Figure 3(d) that represents the TERT expression of group C was incorrect. Also, the western blot signs of TERT and GADPH in Figure 6(c) were swapped. The authors stated that the original figures of the article are not available. The correct figures are as follows:

Figure 1

Subcellular localization of Par-4 and TERT in diabetes. (a) Western blot analysis of Par-4 and TERT expression in cytoplasmic and nuclear extracts in each group. (b) Apoptosis was detected via TUNEL staining in each group. (c) Immunocytochemistry analysis of Par-4 expression in each group. (d) Immunocytochemistry analysis of TERT expression in each group. (e) Expression of Par-4 and TERT, apoptosis rates, cell survival rates determined with MTT assays, and glucose-stimulated insulin secretion in each group. Compared with the C group (P < 0.05). #Compared with the H group (P < 0.05).

Figure 2

Diabetes activates Par-4 and inhibits TERT and p-Akt, inducing islet β cell apoptosis. (a) Immunocytochemistry analysis of Par-4 and TERT expression in cytoplasmic and nuclear extracts in each group. (b) Apoptosis was detected via TUNEL staining in each group. (c) Western blots of Par-4, TERT, Akt, and p-Akt expression in each group. (d) Expression of Par-4 (A), TERT (B), Akt (C), p-Akt (D), apoptosis rates (E), secretion of Par-4 (F), the HOMA-β index (G), and glucose-stimulated insulin secretion (H) in each group. (e) Signal transduction via the Par-4/TERT-Akt pathway to induce islet β cell apoptosis in diabetes. Compared with the N group (P < 0.05). #Compared with the D group (P < 0.05).