Literature DB >> 32046578

Evaluation of urinary neutrophil gelatinase-associated lipocalin and urinary kidney injury molecule-1 as biomarkers of renal function in cancer patients treated with cisplatin.

Elliyeh Ghadrdan1, Sholeh Ebrahimpour2, Sanambar Sadighi3, Samira Chaibakhsh4,5, Zahra Jahangard-Rafsanjani1.   

Abstract

INTRODUCTION: Cisplatin-associated acute kidney injury (AKI) is the major limitation to the use of cisplatin-based chemotherapy regimens. Serum creatinine as a traditional marker did not increase in a timely enough fashion in AKI patients. Therefore, recently, the novel markers such as neutrophil gelatinase-associated lipocalin (NGAL) and kidney injury molecule-1 (KIM-1) were considered for early detection of AKI. The aim of this study was to compare the sensitivity and specificity of urinary NGAL and KIM-1 with serum creatinine in cisplatin related AKI.
METHODS: Patients ≥18 years with solid tumors who received cisplatin-based chemotherapy were included. Urine samples were collected 0, 6 and 24 h after cisplatin infusion and the urinary NGAL, KIM-1, and creatinine concentrations were evaluated. NGAL and KIM-1 concentrations were adjusted based on urine creatinine to eliminate hydration effects. Serum creatinine levels were assessed at the base and 72 h after cisplatin administration.
RESULTS: Seven out of the 35 recruited patients (20%) suffered from AKI defined by Acute Kidney Injury Network criteria. In AKI patients, the ratio of urinary KIM-1-creatinine at 24 h compared to baseline (24 h/baseline) and NGAL-creatinine 24 h/baseline were significantly higher than those of non-AKI group (p = 0.037 and 0.047 respectively). The area under the receiver-operating characteristic curve for KIM-1-creatinine 24 h/baseline and NGAL-creatinine 24 h/baseline were 0.78 (0.59-0.96, p = 0.032) and 0.77 (0.57-0.97, p = 0.036) respectively.
CONCLUSIONS: Our findings showed that the changes in urinary NGAL-creatinine and KIM-1-creatinine ratios, 24 h after cisplatin administration can be utilized to predict AKI in cisplatin recipients.

Entities:  

Keywords:  Acute kidney injury; cisplatin; kidney injury molecule-1; neutrophil gelatinase-associated lipocalin

Mesh:

Substances:

Year:  2020        PMID: 32046578     DOI: 10.1177/1078155220901756

Source DB:  PubMed          Journal:  J Oncol Pharm Pract        ISSN: 1078-1552            Impact factor:   1.809


  4 in total

1.  Urine Neutrophil Gelatinase-Associated Lipocalin and Kidney Injury Molecule-1 to Detect Pediatric Cisplatin-Associated Acute Kidney Injury.

Authors:  Kelly R McMahon; Hayton Chui; Shahrad Rod Rassekh; Kirk R Schultz; Tom D Blydt-Hansen; Cherry Mammen; Maury Pinsk; Geoffrey D E Cuvelier; Bruce C Carleton; Ross T Tsuyuki; Colin J D Ross; Prasad Devarajan; Louis Huynh; Mariya Yordanova; Frédérik Crépeau-Hubert; Stella Wang; Vedran Cockovski; Ana Palijan; Michael Zappitelli
Journal:  Kidney360       Date:  2021-11-03

2.  Kidney Damage and Stress Biomarkers for Early Identification of Drug-Induced Kidney Injury: A Systematic Review.

Authors:  Ravi J Desai; Christina L Kazarov; Adrian Wong; Sandra L Kane-Gill
Journal:  Drug Saf       Date:  2022-07-13       Impact factor: 5.228

3.  Early biomarkers of nephrotoxicity associated with the use of anti-VEGF drugs.

Authors:  Natalia Chebotareva; Katerina Grechukhina; Valerie Mcdonnell; Lyudmila Zhukova; Tatyana Krasnova
Journal:  Biomed Rep       Date:  2022-04-07

4.  Possible protective effect of pantoprazole against cisplatin-induced nephrotoxicity in head and neck cancer patients: a randomized controlled trial.

Authors:  Eman Ghonaim; Sahar El-Haggar; Suzy Gohar
Journal:  Med Oncol       Date:  2021-08-06       Impact factor: 3.064

  4 in total

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