Literature DB >> 32042765

The genetic contribution of HLA-E*01:03 and HLA-E*01:03-G*01:01 to Posner-Schlossman syndrome in southern Chinese.

Xiaosheng Huang1,2, Yunping Xu3, Wenchieh Chen2, Tianhui Zhu1,2, Liumei He3, Songxing Wang3, Shiming Peng1,2, Shaoyi Mei1,2, Yan Wang2, Jun Zhao1,2.   

Abstract

BACKGROUND: The polymorphisms of classical HLA-Ia and HLA-II loci have been associated with Posner-Schlossman syndrome (PSS) in the southern Chinese population. However, the associations of non-classical HLA-Ib (e.g., HLA-E and HLA-G) loci with PSS have not been reported for in the southern Chinese population. This study aimed to evaluate the associations of the HLA-E and HLA-G loci with PSS in a southern Chinese Han population group.
METHODS: Ninety-seven unrelated patients with PSS and 90 ethnically matched control subjects were recruited from the Shenzhen Eye Hospital in China. The full-length sequences of HLA-E and HLA-G genes were amplified by long-range high-fidelity PCR, and the third exon of the HLA-E gene and the coding region of the HLA-G gene were sequenced.
RESULTS: The allele frequency of HLA-E*01:03 in patients with PSS was significantly higher than that in the control group (P=0.017, corrected P=0.034, OR =1.66). The genotype frequencies of HLA-E*01:01/01:03 and HLA-E*01:03/01:03 in the PSS group were significantly higher than that in the control group (P=0.027, OR =2.62; P=0.011, OR =3.05; respectively). There were no significant differences in the frequency of HLA-G alleles and genotypes between the two groups (all P>0.05). The haplotype frequency of HLA-E*01:03-G*01:01 in the PSS group was significantly higher than that in the control group (P=0.019, OR =1.63), although this association did not survive the Bonferroni correction (corrected P=0.13).
CONCLUSIONS: This study proved for the first time that HLA-E*01:03 and HLA-E*01:03-G*01:01 might be risk factors for PSS. 2019 Annals of Translational Medicine. All rights reserved.

Entities:  

Keywords:  Chinese; HLA-E; HLA-G; Posner-Schlossman syndrome (PSS); genetic association

Year:  2019        PMID: 32042765      PMCID: PMC6989981          DOI: 10.21037/atm.2019.11.70

Source DB:  PubMed          Journal:  Ann Transl Med        ISSN: 2305-5839


  33 in total

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