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Literature DB >> 31996035

The crosstalk between hypoxia-inducible factor-1α and microRNAs in acute kidney injury.

Zhiyu Wang¹, Wen Zhang¹.

Abstract

Entities: Disease

Keywords: Acute kidney injury; biomarkers; hypoxia; hypoxia-inducible factor-1α; ischemia; microRNAs

Mesh：

Substances：

Year: 2020 PMID： 31996035 PMCID： PMC7082886 DOI： 10.1177/1535370220902696

Source DB: PubMed Journal: Exp Biol Med (Maywood) ISSN： 1535-3699

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93 in total

1. Factor inhibiting HIF limits the expression of hypoxia-inducible genes in podocytes and distal tubular cells.

Authors: Johannes Schödel; Daniela Bohr; Bernd Klanke; Gunnar Schley; Ursula Schlötzer-Schrehardt; Christina Warnecke; Armin Kurtz; Kerstin Amann; Kai-Uwe Eckardt; Carsten Willam
Journal: Kidney Int Date: 2010-08-18 Impact factor: 10.612

Review 2. Argonaute proteins: key players in RNA silencing.

Authors: Gyorgy Hutvagner; Martin J Simard
Journal: Nat Rev Mol Cell Biol Date: 2008-01 Impact factor: 94.444

3. Preischemic targeting of HIF prolyl hydroxylation inhibits fibrosis associated with acute kidney injury.

Authors: Pinelopi P Kapitsinou; Jonathan Jaffe; Mark Michael; Christina E Swan; Kevin J Duffy; Connie L Erickson-Miller; Volker H Haase
Journal: Am J Physiol Renal Physiol Date: 2012-01-18

4. MicroRNA-494 promotes cyclosporine-induced nephrotoxicity and epithelial to mesenchymal transition by inhibiting PTEN.

Authors: J Yuan; C J Benway; J Bagley; J Iacomini
Journal: Am J Transplant Date: 2015-04-06 Impact factor: 8.086

5. MicroRNA-140-5p attenuated oxidative stress in Cisplatin induced acute kidney injury by activating Nrf2/ARE pathway through a Keap1-independent mechanism.

Authors: Weitang Liao; Zongjie Fu; Yanfang Zou; Dan Wen; Hongkun Ma; Fangfang Zhou; Yongxi Chen; Mingjun Zhang; Wen Zhang
Journal: Exp Cell Res Date: 2017-09-18 Impact factor: 3.905

6. MicroRNA-21 Is Required for Local and Remote Ischemic Preconditioning in Multiple Organ Protection Against Sepsis.

Authors: Ping Jia; Xiaoli Wu; Yan Dai; Jie Teng; Yi Fang; Jiachang Hu; Jianzhou Zou; Mingyu Liang; Xiaoqiang Ding
Journal: Crit Care Med Date: 2017-07 Impact factor: 7.598

7. Induction of microRNA-17-5p by p53 protects against renal ischemia-reperfusion injury by targeting death receptor 6.

Authors: Jielu Hao; Qingqing Wei; Shuqin Mei; Lin Li; Yunchao Su; Changlin Mei; Zheng Dong
Journal: Kidney Int Date: 2016-09-09 Impact factor: 10.612

8. Differentially expressed miRNAs in sepsis-induced acute kidney injury target oxidative stress and mitochondrial dysfunction pathways.

Authors: Qin-Min Ge; Chun-Mei Huang; Xiang-Yang Zhu; Fan Bian; Shu-Ming Pan
Journal: PLoS One Date: 2017-03-15 Impact factor: 3.240

The crosstalk between hypoxia-inducible factor-1α and microRNAs in acute kidney injury.

1. Factor inhibiting HIF limits the expression of hypoxia-inducible genes in podocytes and distal tubular cells.

Review 2. Argonaute proteins: key players in RNA silencing.

3. Preischemic targeting of HIF prolyl hydroxylation inhibits fibrosis associated with acute kidney injury.

4. MicroRNA-494 promotes cyclosporine-induced nephrotoxicity and epithelial to mesenchymal transition by inhibiting PTEN.

5. MicroRNA-140-5p attenuated oxidative stress in Cisplatin induced acute kidney injury by activating Nrf2/ARE pathway through a Keap1-independent mechanism.

6. MicroRNA-21 Is Required for Local and Remote Ischemic Preconditioning in Multiple Organ Protection Against Sepsis.

7. Induction of microRNA-17-5p by p53 protects against renal ischemia-reperfusion injury by targeting death receptor 6.

8. Differentially expressed miRNAs in sepsis-induced acute kidney injury target oxidative stress and mitochondrial dysfunction pathways.

9. A circulating miRNA signature for early diagnosis of acute kidney injury following acute myocardial infarction.

10. Circulating MicroRNA-188, -30a, and -30e as Early Biomarkers for Contrast-Induced Acute Kidney Injury.

1. Analysis of microRNA Expression after Glutamine Intervention in Acute Renal Ischemia-Reperfusion Injury.

2. Renal biomarkers of acute kidney injury in response to increasing intermittent hypoxia episodes in the neonatal rat.