Literature DB >> 31993871

Developing an objective evaluating system to quantify the degree of upper limb movement impairment in patients with severe Friedreich's ataxia.

Giuseppe Arcuria1, Christian Marcotulli2, Raffaele Amuso3, Giuliano Dattilo4, Claudio Galasso2, Francesco Pierelli5, Carlo Casali2.   

Abstract

BACKGROUND: The use of standardized tools and objective measurements is essential to test the effectiveness of new drugs or rehabilitative protocols. Friedreich's ataxia (FRDA) patients with severe disease are often unable to perform the quantitative measurement tests currently used. AIM: The purpose of our study was to develop an easy-to-use application, for touchscreen devices, able to quantify the degree of upper limb movement impairment in patients with severe Friedreich's ataxia. The APP, which we named "Twelve-Red-Squares App-Coo-Test" (12-RSACT), assesses the upper limb ataxia by measuring the test execution time.
METHODS: All patients were clinically evaluated using the Composite Cerebellar Functional Severity (CCFS) and the Scale for the Assessment and Rating of Ataxia (SARA). We recruited 92 healthy subjects and 36 FRDA patients with a SARA mean value of 28.8.1 ± 8.2. All participants in our study underwent upper limb movement assessment using the new 12-RSACT, the Click Test, and a well-established system, i.e., the Nine-Hole Peg Test (9HPT).
RESULTS: We observed a strong linear correlation between the measurements obtained with the 12-RSACT and those obtained with 9HPT, Click Test, CCFS, and SARA. The 12-RSACT was characterized by excellent internal consistency and intra-rater and test-retest reliability. The minimal detectable change (MDC%) was excellent too. Additionally, the 12-RSACT turned out to be faster and easier to perform compared with the 9HPT.
CONCLUSION: The 12-RSACT is an inexpensive test and is easy to use, which can be administered quickly. Therefore, 12-RSACT is a promising tool to assess the upper limb ataxia in FRDA patients and even those with severe diseases.

Entities:  

Keywords:  Friedreich’s ataxia; Quantitative measurements; SARA; Touchscreen application; Upper limb movement impairment

Mesh:

Year:  2020        PMID: 31993871     DOI: 10.1007/s10072-020-04249-0

Source DB:  PubMed          Journal:  Neurol Sci        ISSN: 1590-1874            Impact factor:   3.307


  39 in total

Review 1.  The complex clinical and genetic classification of inherited ataxias. II. Autosomal recessive ataxias.

Authors:  S Di Donato; C Gellera; C Mariotti
Journal:  Neurol Sci       Date:  2001-06       Impact factor: 3.307

2.  Ataxia with isolated vitamin E deficiency: a treatable neurologic disorder resembling Friedreich's ataxia.

Authors:  A Federico
Journal:  Neurol Sci       Date:  2004-07       Impact factor: 3.307

3.  GIFT-1, a phase IIa clinical trial to test the safety and efficacy of IFNγ administration in FRDA patients.

Authors:  Christian Marcotulli; Silvia Fortuni; Gaetano Arcuri; Barbara Tomassini; Luca Leonardi; Francesco Pierelli; Roberto Testi; Carlo Casali
Journal:  Neurol Sci       Date:  2015-11-30       Impact factor: 3.307

4.  G130V, a common FRDA point mutation, appears to have arisen from a common founder.

Authors:  M B Delatycki; M Knight; M Koenig; M Cossée; R Williamson; S M Forrest
Journal:  Hum Genet       Date:  1999-10       Impact factor: 4.132

5.  Clinical and genetic study of Friedreich ataxia in an Australian population.

Authors:  M B Delatycki; D B Paris; R J Gardner; G A Nicholson; N Nassif; E Storey; J C MacMillan; V Collins; R Williamson; S M Forrest
Journal:  Am J Med Genet       Date:  1999-11-19

6.  A wearable proprioceptive stabilizer for rehabilitation of limb and gait ataxia in hereditary cerebellar ataxias: a pilot open-labeled study.

Authors:  Luca Leonardi; Maria Gabriella Aceto; Christian Marcotulli; Giuseppe Arcuria; Mariano Serrao; Francesco Pierelli; Paolo Paone; Alessandro Filla; Alessandro Roca; Carlo Casali
Journal:  Neurol Sci       Date:  2016-12-31       Impact factor: 3.307

7.  Absence of aprataxin gene mutations in a Greek cohort with sporadic early onset ataxia and normal GAA triplets in frataxin gene.

Authors:  C Daiou; K Christodoulou; G Xiromerisiou; M Panas; E Dardiotis; A Kladi; M Speletas; G Ntaios; A Papadimitriou; A Germenis; Georgios M Hadjigeorgiou
Journal:  Neurol Sci       Date:  2009-12-02       Impact factor: 3.307

Review 8.  The pathogenesis of Friedreich ataxia and the structure and function of frataxin.

Authors:  Massimo Pandolfo; Annalisa Pastore
Journal:  J Neurol       Date:  2009-03       Impact factor: 4.849

Review 9.  Autosomal recessive ataxias: 20 types, and counting.

Authors:  Emília Katiane Embiruçu; Marcília Lima Martyn; David Schlesinger; Fernando Kok
Journal:  Arq Neuropsiquiatr       Date:  2009-12       Impact factor: 1.420

10.  Friedreich's ataxia: point mutations and clinical presentation of compound heterozygotes.

Authors:  M Cossée; A Dürr; M Schmitt; N Dahl; P Trouillas; P Allinson; M Kostrzewa; A Nivelon-Chevallier; K H Gustavson; A Kohlschütter; U Müller; J L Mandel; A Brice; M Koenig; F Cavalcanti; A Tammaro; G De Michele; A Filla; S Cocozza; M Labuda; L Montermini; J Poirier; M Pandolfo
Journal:  Ann Neurol       Date:  1999-02       Impact factor: 10.422

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