Literature DB >> 31976583

Fine-Mapping of SNCA in Rapid Eye Movement Sleep Behavior Disorder and Overt Synucleinopathies.

Lynne Krohn1,2, Richard Y J Wu1,3, Karl Heilbron4, Jennifer A Ruskey2,5, Sandra B Laurent2,5, Cornelis Blauwendraat6, Armaghan Alam1,2, Isabelle Arnulf7, Michele T M Hu8,9, Yves Dauvilliers10, Birgit Högl11, Mathias Toft12,13, Kari Anne Bjørnarå12, Ambra Stefani11, Evi Holzknecht11, Christelle Charley Monaca14, Beatriz Abril15, Giuseppe Plazzi16,17, Elena Antelmi16,17, Luigi Ferini-Strambi18, Peter Young19, Anna Heidbreder19, Valérie Cochen De Cock20,21, Brit Mollenhauer22,23, Friederike Sixel-Döring22,23, Claudia Trenkwalder22,23, Karel Sonka24, David Kemlink24, Michela Figorilli25, Monica Puligheddu25, Femke Dijkstra26,27, Mineke Viaene26,27, Wolfang Oertel28, Marco Toffoli29,30, Gian Luigi Gigli31,32, Mariarosaria Valente29,31, Jean-François Gagnon33,34, Mike A Nalls35, Andrew B Singleton6, Alex Desautels33,36, Jacques Y Montplaisir33,37, Paul Cannon4, Owen A Ross38, Bradley F Boeve39, Nicolas Dupré40,41, Edward A Fon2,5, Ronald B Postuma2,5,33, Lasse Pihlstrøm12, Guy A Rouleau1,2,5, Ziv Gan-Or1,2,5.   

Abstract

OBJECTIVE: Rapid eye movement sleep behavior disorder (RBD) is a prodromal synucleinopathy, as >80% will eventually convert to overt synucleinopathy. We performed an in-depth analysis of the SNCA locus to identify RBD-specific risk variants.
METHODS: Full sequencing and genotyping of SNCA was performed in isolated/idiopathic RBD (iRBD, n = 1,076), Parkinson disease (PD, n = 1,013), dementia with Lewy bodies (DLB, n = 415), and control subjects (n = 6,155). The iRBD cases were diagnosed with RBD prior to neurodegeneration, although some have since converted. A replication cohort from 23andMe of PD patients with probable RBD (pRBD) was also analyzed (n = 1,782 cases; n = 131,250 controls). Adjusted logistic regression models and meta-analyses were performed. Effects on conversion rate were analyzed in 432 RBD patients with available data using Kaplan-Meier survival analysis.
RESULTS: A 5'-region SNCA variant (rs10005233) was associated with iRBD (odds ratio [OR] = 1.43, p = 1.1E-08), which was replicated in pRBD. This variant is in linkage disequilibrium (LD) with other 5' risk variants across the different synucleinopathies. An independent iRBD-specific suggestive association (rs11732740) was detected at the 3' of SNCA (OR = 1.32, p = 4.7E-04, not statistically significant after Bonferroni correction). Homozygous carriers of both iRBD-specific SNPs were at highly increased risk for iRBD (OR = 5.74, p = 2E-06). The known top PD-associated variant (3' variant rs356182) had an opposite direction of effect in iRBD compared to PD.
INTERPRETATION: There is a distinct pattern of association at the SNCA locus in RBD as compared to PD, with an opposite direction of effect at the 3' of SNCA. Several 5' SNCA variants are associated with iRBD and with pRBD in overt synucleinopathies. ANN NEUROL 2020;87:584-598.
© 2020 American Neurological Association.

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Year:  2020        PMID: 31976583      PMCID: PMC8025046          DOI: 10.1002/ana.25687

Source DB:  PubMed          Journal:  Ann Neurol        ISSN: 0364-5134            Impact factor:   11.274


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