Literature DB >> 31965202

[Extended understanding of pathogenesis and treatment of contact allergy].

Philipp R Esser1, Stefan F Martin2.   

Abstract

BACKGROUND: While the pathogenesis of contact allergy in recent years has increasingly focused on the mechanisms of the innate immune response, valid therapeutic options are still lacking. AIMS: This article intends to shed light on the background of contact allergy development as well as possible risk factors and to highlight potential new therapeutic options.
MATERIALS AND METHODS: Allergic contact dermatitis (ACD) as well as the sensitization and trigger phase, potential risk factors as well as the therapy options including (current) PubMed-listed literature are described.
RESULTS: Inflammation plays a central role in ACD. The innate immune system responds to contact allergens as well as to infection. Elucidation of the mechanisms will enable a targeted therapeutic intervention in the future.
CONCLUSION: Although there is still a need for research, many parts of the contact allergy pathogenesis are now better understood. In particular, the essential role of the innate immune response not only for the sensitization but also for the elicitation phase seems to be established. Implementation of today's knowledge into new therapeutic approaches and their application testing remains important and exciting.

Entities:  

Keywords:  Allergic contact dermatitis; Haptens; Inflammation; Innate immune response; Risk factors

Year:  2020        PMID: 31965202     DOI: 10.1007/s00105-019-04527-9

Source DB:  PubMed          Journal:  Hautarzt        ISSN: 0017-8470            Impact factor:   0.751


  42 in total

1.  Age-specific profiling of cutaneous allergy at high temporal resolution suggests age-related alterations in regulatory immune function.

Authors:  Magnus David Lynch; John Paul McFadden; Jonathan Michael White; Piu Banerjee; Ian Richard White
Journal:  J Allergy Clin Immunol       Date:  2017-06-09       Impact factor: 10.793

2.  Nickel acts as an adjuvant during cobalt sensitization.

Authors:  Charlotte Menné Bonefeld; Morten Milek Nielsen; Marie T Vennegaard; Jeanne Duus Johansen; Carsten Geisler; Jacob P Thyssen
Journal:  Exp Dermatol       Date:  2015-03       Impact factor: 3.960

3.  Metal allergens nickel and cobalt facilitate TLR4 homodimerization independently of MD2.

Authors:  Badrinarayanan Raghavan; Stefan F Martin; Philipp R Esser; Matthias Goebeler; Marc Schmidt
Journal:  EMBO Rep       Date:  2012-10-12       Impact factor: 8.807

4.  Mast cells are key promoters of contact allergy that mediate the adjuvant effects of haptens.

Authors:  Anne Dudeck; Jan Dudeck; Julia Scholten; Anke Petzold; Sangeetha Surianarayanan; Anja Köhler; Katrin Peschke; David Vöhringer; Claudia Waskow; Thomas Krieg; Werner Müller; Ari Waisman; Karin Hartmann; Matthias Gunzer; Axel Roers
Journal:  Immunity       Date:  2011-06-24       Impact factor: 31.745

5.  Association between TNFA-308 G/A polymorphism and sensitization to para-phenylenediamine: a case-control study.

Authors:  B Blömeke; R Brans; H Dickel; T Bruckner; S Erdmann; M Heesen; H F Merk; P-J Coenraads
Journal:  Allergy       Date:  2008-03-29       Impact factor: 13.146

6.  Lack of the purinergic receptor P2X(7) results in resistance to contact hypersensitivity.

Authors:  Felix C Weber; Philipp R Esser; Tobias Müller; Jayanthi Ganesan; Patrizia Pellegatti; Markus M Simon; Robert Zeiser; Marco Idzko; Thilo Jakob; Stefan F Martin
Journal:  J Exp Med       Date:  2010-11-08       Impact factor: 14.307

7.  Skin sensitization induced Langerhans' cell mobilization: variable requirements for tumour necrosis factor-α.

Authors:  Laura H Eaton; Ruth A Roberts; Ian Kimber; Rebecca J Dearman; Aleksandra Metryka
Journal:  Immunology       Date:  2015-01       Impact factor: 7.397

Review 8.  Guideline contact dermatitis: S1-Guidelines of the German Contact Allergy Group (DKG) of the German Dermatology Society (DDG), the Information Network of Dermatological Clinics (IVDK), the German Society for Allergology and Clinical Immunology (DGAKI), the Working Group for Occupational and Environmental Dermatology (ABD) of the DDG, the Medical Association of German Allergologists (AeDA), the Professional Association of German Dermatologists (BVDD) and the DDG.

Authors:  Jochen Brasch; Detlef Becker; Werner Aberer; Andreas Bircher; Birger Kränke; Kirsten Jung; Bernhard Przybilla; Tilo Biedermann; Thomas Werfel; Swen Malte John; Peter Elsner; Thomas Diepgen; Axel Trautmann; Hans F Merk; Thomas Fuchs; Axel Schnuch
Journal:  Allergo J Int       Date:  2014

9.  Neutrophils are required for both the sensitization and elicitation phase of contact hypersensitivity.

Authors:  Felix C Weber; Tamás Németh; Janka Z Csepregi; Anne Dudeck; Axel Roers; Béla Ozsvári; Eva Oswald; László G Puskás; Thilo Jakob; Attila Mócsai; Stefan F Martin
Journal:  J Exp Med       Date:  2014-12-15       Impact factor: 14.307

10.  Toll-like receptor and IL-12 signaling control susceptibility to contact hypersensitivity.

Authors:  Stefan F Martin; Jan C Dudda; Eva Bachtanian; Annalisa Lembo; Stefanie Liller; Christoph Dürr; Markus M Heimesaat; Stefan Bereswill; György Fejer; Ralitsa Vassileva; Thilo Jakob; Nikolaus Freudenberg; Christian C Termeer; Caroline Johner; Chris Galanos; Marina A Freudenberg
Journal:  J Exp Med       Date:  2008-08-25       Impact factor: 14.307

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  2 in total

Review 1. 

Authors:  Jörg Kleine-Tebbe; Richard Brans; Uta Jappe
Journal:  Allergo J       Date:  2022-03-21

2.  Nickel: Intrinsic Skin Sensitization Potency and Relation to Prevalence of Contact Allergy.

Authors:  David Basketter
Journal:  Dermatitis       Date:  2021 Mar-Apr 01       Impact factor: 4.867

  2 in total

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