Literature DB >> 31953125

Biochemical characterization of tirabrutinib and other irreversible inhibitors of Bruton's tyrosine kinase reveals differences in on - and off - target inhibition.

Albert Liclican1, Loredana Serafini1, Weimei Xing1, Gregg Czerwieniec1, Bart Steiner1, Ting Wang1, Katherine M Brendza1, Justin D Lutz1, Kathleen S Keegan1, Adrian S Ray1, Brian E Schultz1, Roman Sakowicz1, Joy Y Feng2.   

Abstract

BACKGROUND: Bruton's tyrosine kinase (BTK) is a key component of the B-cell receptor (BCR) pathway and a clinically validated target for small molecule inhibitors such as ibrutinib in the treatment of B-cell malignancies. Tirabrutinib (GS-4059/ONO-4059) is a selective, once daily, oral BTK inhibitor with clinical activity against many relapsed/refractory B-cell malignancies.
METHODS: Covalent binding of tirabrutinib to BTK Cys-481 was assessed by LC-MSMS analysis of BTK using compound as a variable modification search parameter. Inhibition potency of tirabrutinib, ibrutinib, acalabrutinib, and spebrutinib against BTK and related kinases was studied in a dose-dependent manner either after a fixed incubation time (as used in conventional IC50 studies) or following a time course where inactivation kinetics were measured.
RESULTS: Tirabrutinib irreversibly and covalently binds to BTK Cys-481. The inactivation efficiency kinact/Ki was measured and used to calculate selectivity among different kinases for each of the four inhibitors studied. Tirabrutinib showed a kinact/Ki value of 2.4 ± 0.6 × 104 M-1 s-1 for BTK with selectivity against important off-targets.
CONCLUSIONS: For the BTK inhibitors tested in this study, analysis of the inactivation kinetics yielded a more accurate measurement of potency and selectivity than conventional single-time point inhibition measurements. Subtle but clear differences were identified between clinically tested BTK inhibitors which may translate into differentiated clinical efficacy and safety. GENERAL SIGNIFICANCE: This is the first study that offers a detailed side-by-side comparison of four clinically-relevant BTK inhibitors with respect to their inactivation of BTK and related kinases.
Copyright © 2020 The Authors. Published by Elsevier B.V. All rights reserved.

Entities:  

Keywords:  BTK inhibitor; Bruton's tyrosine kinase; Ibrutinib; Inactivation kinetics; Tirabrutinib

Year:  2020        PMID: 31953125     DOI: 10.1016/j.bbagen.2020.129531

Source DB:  PubMed          Journal:  Biochim Biophys Acta Gen Subj        ISSN: 0304-4165            Impact factor:   3.770


  20 in total

1.  Photoinduced Covalent Irreversible Inactivation of Proline Dehydrogenase by S-Heterocycles.

Authors:  Ashley C Campbell; Austin R Prater; Alexandra N Bogner; Thomas P Quinn; Kent S Gates; Donald F Becker; John J Tanner
Journal:  ACS Chem Biol       Date:  2021-09-20       Impact factor: 5.100

2.  Experience with nasogastric tube administration of tirabrutinib in the treatment of an elderly patient with primary central nervous system lymphoma.

Authors:  Hiromasa Yoshioka; Takeshi Okuda; Takayuki Nakao; Mitsugu Fujita; Jun C Takahashi
Journal:  Int Cancer Conf J       Date:  2021-06-05

3.  Discovery of a Potent and Selective Covalent Inhibitor of Bruton's Tyrosine Kinase with Oral Anti-Inflammatory Activity.

Authors:  Mark S Tichenor; John J M Wiener; Navin L Rao; Charlotte Pooley Deckhut; J Kent Barbay; Kevin D Kreutter; Genesis M Bacani; Jianmei Wei; Leon Chang; Heather E Murrey; Weixue Wang; Kay Ahn; Michael Huber; Elizabeth Rex; Kevin J Coe; JieJun Wu; Mark Seierstad; Scott D Bembenek; Kristi A Leonard; Alec D Lebsack; Jennifer D Venable; James P Edwards
Journal:  ACS Med Chem Lett       Date:  2021-04-05       Impact factor: 4.345

4.  Detection of an enzyme isomechanism by means of the kinetics of covalent inhibition.

Authors:  S A Adediran; Michael J Morrison; R F Pratt
Journal:  Biochim Biophys Acta Proteins Proteom       Date:  2021-06-02       Impact factor: 4.125

5.  Semi-Mechanistic PK/PD Modeling and Simulation of Irreversible BTK Inhibition to Support Dose Selection of Tirabrutinib in Subjects with RA.

Authors:  Amy Meng; Rita Humeniuk; Juliane M Jürgensmeier; Chia-Hsiang Hsueh; Franziska Matzkies; Ethan Grant; Hoa Truong; Andrew N Billin; Helen Yu; Joy Feng; Ellen Kwan; Thomas Tarnowski; Cara H Nelson
Journal:  Clin Pharmacol Ther       Date:  2021-10-27       Impact factor: 6.903

6.  A multicenter, open-label, phase II study of tirabrutinib (ONO/GS-4059) in patients with Waldenström's macroglobulinemia.

Authors:  Naohiro Sekiguchi; Shinya Rai; Wataru Munakata; Kenshi Suzuki; Hiroshi Handa; Hirohiko Shibayama; Tomoyuki Endo; Yasuhito Terui; Noriko Iwaki; Noriko Fukuhara; Hiro Tatetsu; Shinsuke Iida; Takayuki Ishikawa; Ryota Shiibashi; Koji Izutsu
Journal:  Cancer Sci       Date:  2020-07-20       Impact factor: 6.716

7.  Mechanism of covalent binding of ibrutinib to Bruton's tyrosine kinase revealed by QM/MM calculations.

Authors:  Angus T Voice; Gary Tresadern; Rebecca M Twidale; Herman van Vlijmen; Adrian J Mulholland
Journal:  Chem Sci       Date:  2021-01-28       Impact factor: 9.825

Review 8.  Reining in BTK: Interdomain Interactions and Their Importance in the Regulatory Control of BTK.

Authors:  Lauren E Kueffer; Raji E Joseph; Amy H Andreotti
Journal:  Front Cell Dev Biol       Date:  2021-06-23

9.  Two-year outcomes of tirabrutinib monotherapy in Waldenström's macroglobulinemia.

Authors:  Naohiro Sekiguchi; Shinya Rai; Wataru Munakata; Kenshi Suzuki; Hiroshi Handa; Hirohiko Shibayama; Tomoyuki Endo; Yasuhito Terui; Noriko Iwaki; Noriko Fukuhara; Hiro Tatetsu; Shinsuke Iida; Takayuki Ishikawa; Daisuke Iguchi; Koji Izutsu
Journal:  Cancer Sci       Date:  2022-04-06       Impact factor: 6.518

Review 10.  Ibrutinib in Gynecological Malignancies and Breast Cancer: A Systematic Review.

Authors:  Julian Matthias Metzler; Laurin Burla; Daniel Fink; Patrick Imesch
Journal:  Int J Mol Sci       Date:  2020-06-10       Impact factor: 5.923

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