Literature DB >> 31933901

Down-regulated miR-21 promotes learning-memory recovery after brain injury.

You-Meng Wang1, Zhen Song2, Yi Qu3, Li-Qun Lu1.   

Abstract

BACKGROUND: MicroRNA is anendogenous non-coding single strand RNA which consists of 22 nt. It post-transcriptionally regulates gene expression and development. MicroRNA 21 plays an important role in repairing injured brain tissues. Thus, in this research, we explored the function of miR-21 in learning-memory recovery after brain injury.
METHOD: 3 days old newborn SD rats were separated into three groups: Sham operation group (Sham), inflammation-sensitized hypoxic-ischemic brain injury (LPS+HI) group and miR-21 inhibitor group. 28 days later, the learning and memory capability was assayed by water maze. H&E staining and Nissl's staining were used to assay pathologic changes and TUNEL was used to assay neuron apoptosis in brain tissue.
RESULTS: Water maze assay showed that the capability of positioning navigation in the IH-HI group was worse than in the Sham group and miR-21 inhibition group, and the Sham group wass better than miR-21 group. Both of the comparisons had statistical significance (P < 0.05). H&E staining in the sham group showed that the neurons were arranged well in hippocampus. In LPS+HI group, some neurons in hippocampus had vacuolar degeneration and the neurons were not well arranged well. In the hippocampus of miR-21 inhibitor group, the neuron cell layers were decreased but the neurons were arranged better than in the LPS+HI group. Nissl's staining in LPS+HI group showed neuronal edema, neurons decreased, and Nissl's bodies decreased in the cytoplasm compared with the sham group. However, compared with the LPS+HI group, Nissl's staining in miR-21 inhibitor group showed that the neuronal edema was alleviated and neurons were better arranged. TUNEL assay showed that the apoptosis rate of LPS+HI group was higher than in the miR-21 inhibitor group and miR-21 inhibitor group was higher than the sham group.
CONCLUSION: Down-regulated miR-21 can alleviate LPS+HI injury in the brain. IJCEP
Copyright © 2019.

Entities:  

Keywords:  Brain injury; learning-memory recovery; miR-21

Year:  2019        PMID: 31933901      PMCID: PMC6945147     

Source DB:  PubMed          Journal:  Int J Clin Exp Pathol        ISSN: 1936-2625


  6 in total

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Journal:  Mol Immunol       Date:  2018-06-06       Impact factor: 4.407

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5.  TNFR1-JNK signaling is the shared pathway of neuroinflammation and neurovascular damage after LPS-sensitized hypoxic-ischemic injury in the immature brain.

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Authors:  Xiaodong Ma; Daniel J Conklin; Fenge Li; Zhongping Dai; Xiang Hua; Yan Li; Zijun Y Xu-Monette; Ken H Young; Wei Xiong; Marcin Wysoczynski; Srinivas D Sithu; Sanjay Srivastava; Aruni Bhatnagar; Yong Li
Journal:  Nat Commun       Date:  2015-05-20       Impact factor: 14.919

  6 in total
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Journal:  Biomedicines       Date:  2021-12-30
  1 in total

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