Correction to: miR199a-3p regulates P53 by targeting CABLES1 in mouse cardiac c-kit+ cells to promote proliferation and inhibit apoptosis through a negative feedback loop.

After publication of our article [1] we became aware that there were errors in Fig. 5b and Fig. 6c, namely that the immunofluorescence of EDU-positive cells of the CABLES1 transfection group in Fig. 5b (panel 2) and the cell cycle distribution of the combination group (treatment with the antimiR199a-3p and shRNA-CABLES1) in Fig. 6c (panel 3) were incorrectly presented.

Correction to: Stem Cell Res Ther (2017) 8:127

https://doi.org/10.1186/s13287-017-0515-4

After publication of our article [1] we became aware that there were errors in Fig. 5b and Fig. 6c, namely that the immunofluorescence of EDU-positive cells of the CABLES1 transfection group in Fig. 5b (panel 2) and the cell cycle distribution of the combination group (treatment with the antimiR199a-3p and shRNA-CABLES1) in Fig. 6c (panel 3) were incorrectly presented. These errors do not affect the discussion or conclusions in the article. The correct versions of Figs. 5 and 6 are shown below. We apologize to the journal and to readers for this error.

Fig. 5

miR199a-3p regulates cell proliferation and apoptosis through targeting CABLES1. a Western blot analysis of proteins from cardiac c-kit+ cells transfected with the miR199a-3p and CABLES1 lentiviral vectors (*p < 0.05). The protein profiles were normalized to α-actin. b The cells were stained with EdU and DAPI (*p < 0.05). c Cell cycle distribution of cardiac c-kit+ cells after transfection with the miR199a-3p and CABLES1 lentiviral vectors. d Representative annexin V/PI flow cytometry analysis of cardiac c-kit+ cells and analysis of annexin V+/PI+ cardiac c-kit+ cells by flow cytometry. CABLES1 Cdk5 and Abl enzyme substrate 1, Con control, PI propidium iodide (*p < 0.05)

Fig. 6

miR199a-3p regulates cell proliferation and apoptosis through targeting CABLES1. a Western blot analysis of proteins from cardiac c-kit+ cells transfected with the anti-miR199a-3p and shRNA-CABLES1 lentiviral vectors (*p < 0.05). b The cells were stained with EdU and DAPI (*p < 0.05). c Cell cycle distribution of cardiac c-kit+ cells after transfection with the anti-miR199a-3p and shRNA-CABLES1 lentiviral vectors. d Representative annexin V/PI flow cytometry analysis of cardiac c-kit+ cells and analysis of annexin V+/PI+ cardiac c-kit+ cells by flow cytometry (*p < 0.05). CABLES1 Cdk5 and Abl enzyme substrate 1, Con control, PI propidium iodide