Literature DB >> 31845496

Sodium retention by uPA-plasmin-ENaC in nephrotic syndrome-Authors reply.

Gitte R Hinrichs1, Kathrin Weyer2, Ulla G Friis1, Per Svenningsen1, Ida Katrine Lund3, Rikke Nielsen2, Géraldine Mollet4, Corinne Antignac4,5, Claus Bistrup6,7, Boye L Jensen1, Henrik Birn2,8.   

Abstract

Entities:  

Year:  2020        PMID： 31845496     DOI： 10.1111/apha.13432

Source DB:  PubMed          Journal:  Acta Physiol (Oxf)        ISSN： 1748-1708            Impact factor:   6.311


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  2 in total

1.  Plasminogen deficiency does not prevent sodium retention in a genetic mouse model of experimental nephrotic syndrome.

Authors:  Mengyun Xiao; Bernhard N Bohnert; Hande Aypek; Oliver Kretz; Florian Grahammer; Ute Aukschun; Matthias Wörn; Andrea Janessa; Daniel Essigke; Christoph Daniel; Kerstin Amann; Tobias B Huber; Edward F Plow; Andreas L Birkenfeld; Ferruh Artunc
Journal:  Acta Physiol (Oxf)       Date:  2020-06-10       Impact factor: 6.311

2.  Melatonin Attenuates ox-LDL-Induced Endothelial Dysfunction by Reducing ER Stress and Inhibiting JNK/Mff Signaling.

Authors:  Peng Li; Changlian Xie; Jiankai Zhong; Zhongzhou Guo; Kai Guo; Qiuyun Tu
Journal:  Oxid Med Cell Longev       Date:  2021-03-04       Impact factor: 6.543
  2 in total

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