Literature DB >> 31810576

Paroxysmal tonic upgaze: A heterogeneous clinical condition responsive to carbonic anhydrase inhibition.

Annegret Quade1, Anne Thiel2, Ingo Kurth3, Manuel Holtgrewe4, Miriam Elbracht3, Dieter Beule4, Katja Eggermann3, Ute I Scholl5, Martin Häusler6.   

Abstract

BACKGROUND: Paroxysmal tonic upgaze (PTU), defined as an involuntary upward movement of the eyes, has been considered as a benign phenomenon but may also be associated with ataxia and developmental delay.
METHODS: We report eight children with PTU; six of them also exhibiting symptoms of ataxia and/or developmental delay. Treatment with carbonic anhydrase inhibition was offered to children with persisting and/or severe forms.
RESULTS: Whole-exome sequencing and genome-wide array analysis (n = 7) did not reveal mutations in the three known genes associated with PTU (CACNA1A, GRID2, SEPSECS), whereas by MLPA a heterozygous deletion of exon 31 of the CACNA1A gene could be detected in one patient, her mother and two further family members. Further exome and array analysis showed no recurrent variants in potentially novel PTU-related genes in more than one patient. A de novo variant at a highly conserved position in the SIM1 gene was detected in one patient, for which a pathogenic effect could be speculated. Carbonic anhydrase inhibition was started in five children and proved at least partially effective in all of them.
CONCLUSION: Irrespective of the clinical background and the molecular basic mechanism of PTU, therapeutic carbonic anhydrase inhibition was effective in all five children (acetazolamide, n = 3; sultiame, n = 2) who received this treatment.
Copyright © 2019 European Paediatric Neurology Society. Published by Elsevier Ltd. All rights reserved.

Entities:  

Keywords:  Acetazolamide; CACNA1A; Episodic ataxia; Paroxysmal tonic upgaze; SIM1; Sultiame

Year:  2019        PMID: 31810576     DOI: 10.1016/j.ejpn.2019.11.002

Source DB:  PubMed          Journal:  Eur J Paediatr Neurol        ISSN: 1090-3798            Impact factor:   3.140


  3 in total

1.  The complexities of CACNA1A in clinical neurogenetics.

Authors:  Marina P Hommersom; Teije H van Prooije; Maartje Pennings; Meyke I Schouten; Hans van Bokhoven; Erik-Jan Kamsteeg; Bart P C van de Warrenburg
Journal:  J Neurol       Date:  2021-11-22       Impact factor: 6.682

2.  Haploinsufficiency of POU4F1 causes an ataxia syndrome with hypotonia and intention tremor.

Authors:  Bryn D Webb; Anthony Evans; Thomas P Naidich; Lynne M Bird; Sumit Parikh; Meilin Fernandez Garcia; Lindsay B Henderson; Francisca Millan; Yue Si; Kristen J Brennand; Peter Hung; Janet C Rucker; Patricia G Wheeler; Eric E Schadt
Journal:  Hum Mutat       Date:  2021-04-15       Impact factor: 4.700

Review 3.  Clinical and Genetic Overview of Paroxysmal Movement Disorders and Episodic Ataxias.

Authors:  Giacomo Garone; Alessandro Capuano; Lorena Travaglini; Federica Graziola; Fabrizia Stregapede; Ginevra Zanni; Federico Vigevano; Enrico Bertini; Francesco Nicita
Journal:  Int J Mol Sci       Date:  2020-05-20       Impact factor: 5.923

  3 in total

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