Literature DB >> 31792333

Participation of white adipose tissue dysfunction on circulating HDL cholesterol and HDL particle size in apparently healthy humans.

Juan G Juárez-Rojas1, Ivan Torre-Villalvazo2, Aida X Medina-Urrutia1, Juan Reyes-Barrera1, Víctor H Sainz-Escárrega3, Carlos Posadas-Romero1, Alejandro Macías-Cruz1, Esteban Jorge-Galarza4.   

Abstract

OBJECTIVE: To use the combined presence of the elevated insulin resistance index in adipose tissue (Adipo-IR) and low values of adiponectin as a marker of dysfunctional adipose tissue, and to analyze its possible association with low values of high-density lipoprotein cholesterol (HDL-C) and small size of HDL particles. RESEARCH DESIGN AND METHODS: The analysis included 253 subjects with functional adipose tissue and 253 with dysfunctional adipose tissue, considering similar gender, age, and body mass index (BMI). Adipo-IR was considered when index values (free fatty acids × insulin concentrations) were ≥75th percentile. Low levels of adiponectin were considered when concentration in serum was <25th percentile (determined by ELISA). HDL size was estimated by a quantitative validated equation. Small HDL size was considered when values were <25th percentile.
RESULTS: When comparing subjects with functional adipose tissue with those of dysfunctional adipose tissue, the latter had a higher prevalence of low HDL-C (51.4% vs. 64.0%; p = 0.004) and small HDL (56.9% vs. 67.6%; p = 0.009). Multivariate analysis indicated that independently from other metabolic risk factors, dysfunction of adipose tissue is significantly associated with low HDL-C (OR: 1.624 [CI 95%: 1.100-2.397]) and small HDL (OR: 1.462 [CI 95%: 1.000-2.139]). Adding BMI, waist circumference, and subcutaneous or visceral adipose tissue did not modify the association.
CONCLUSIONS: Dysfunction of adipose tissue is associated with a 65 and 50% higher probability of having low HDL-C and small HDL. Identification of dysfunctional adipose tissue could be a useful tool in the clinical setting to prevent the cardiometabolic risk independently from adiposity.

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Year:  2019        PMID: 31792333     DOI: 10.1038/s41366-019-0493-y

Source DB:  PubMed          Journal:  Int J Obes (Lond)        ISSN: 0307-0565            Impact factor:   5.095


  40 in total

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3.  Adipocyte modulation of high-density lipoprotein cholesterol.

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Journal:  Circulation       Date:  2010-03-08       Impact factor: 29.690

Review 4.  Molecular mechanisms underlying metabolic syndrome: the expanding role of the adipocyte.

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Journal:  FASEB J       Date:  2017-07-13       Impact factor: 5.191

Review 5.  Adipose tissue and cholesterol metabolism.

Authors:  B R Krause; A D Hartman
Journal:  J Lipid Res       Date:  1984-02       Impact factor: 5.922

6.  Role of Adipose Tissue Insulin Resistance in the Natural History of Type 2 Diabetes: Results From the San Antonio Metabolism Study.

Authors:  Amalia Gastaldelli; Melania Gaggini; Ralph A DeFronzo
Journal:  Diabetes       Date:  2017-01-04       Impact factor: 9.461

Review 7.  Adipose tissue, inflammation and atherosclerosis.

Authors:  Birgit Gustafson
Journal:  J Atheroscler Thromb       Date:  2010-02-03       Impact factor: 4.928

8.  High density lipoprotein as a protective factor against coronary heart disease. The Framingham Study.

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Journal:  Am J Med       Date:  1977-05       Impact factor: 4.965

9.  Role of dysfunctional adipocytes in cholesterol-induced nonobese metabolic syndrome.

Authors:  Jiung-Pang Huang; Sheng-Chieh Hsu; Yaa-Jyuhn James Meir; Po-Shiuan Hsieh; Chih-Chun Chang; Kuan-Hsing Chen; Jan-Kan Chen; Li-Man Hung
Journal:  J Mol Endocrinol       Date:  2018-03-26       Impact factor: 5.098

10.  Receptor-mediated activation of ceramidase activity initiates the pleiotropic actions of adiponectin.

Authors:  William L Holland; Russell A Miller; Zhao V Wang; Kai Sun; Brian M Barth; Hai H Bui; Kathryn E Davis; Benjamin T Bikman; Nils Halberg; Joseph M Rutkowski; Mark R Wade; Vincent M Tenorio; Ming-Shang Kuo; Joseph T Brozinick; Bei B Zhang; Morris J Birnbaum; Scott A Summers; Philipp E Scherer
Journal:  Nat Med       Date:  2010-12-26       Impact factor: 53.440

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  1 in total

1.  Vitamin D Receptor Is a Sepsis-Susceptibility Gene in Chinese Children.

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Journal:  Med Sci Monit       Date:  2021-10-24
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