| Literature DB >> 31782728 |
Guillermo Luxán1, Jonas Stewen1, Mara E Pitulescu1, Ralf H Adams1,2, Noelia Díaz3, Katsuhiro Kato1, Sathish K Maney1, Anusha Aravamudhan1, Frank Berkenfeld1, Nina Nagelmann4, Hannes Ca Drexler5, Dagmar Zeuschner6, Cornelius Faber4, Hermann Schillers7, Sven Hermann8, John Wiseman9, Juan M Vaquerizas3.
Abstract
The homeostasis of heart and other organs relies on the appropriate provision of nutrients and functional specialization of the local vasculature. Here, we have used mouse genetics, imaging and cell biology approaches to investigate how homeostasis in the adult heart is controlled by endothelial EphB4 and its ligand ephrin-B2, which are known regulators of vascular morphogenesis and arteriovenous differentiation during development. We show that inducible and endothelial cell-specific inactivation of Ephb4 in adult mice is compatible with survival, but leads to rupturing of cardiac capillaries, cardiomyocyte hypertrophy, and pathological cardiac remodeling. In contrast, EphB4 is not required for integrity and homeostasis of capillaries in skeletal muscle. Our analysis of mutant mice and cultured endothelial cells shows that EphB4 controls the function of caveolae, cell-cell adhesion under mechanical stress and lipid transport. We propose that EphB4 maintains critical functional properties of the adult cardiac vasculature and thereby prevents dilated cardiomyopathy-like defects.Entities:
Keywords: blood vessel; cardiomyocyte; developmental biology; endothelial cell; heart; homeostasis; mouse
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Year: 2019 PMID: 31782728 PMCID: PMC6884395 DOI: 10.7554/eLife.45863
Source DB: PubMed Journal: Elife ISSN: 2050-084X Impact factor: 8.140