Literature DB >> 31770102

Congenital hypogonadotropic hypogonadism/Kallmann syndrome is associated with statural gain in both men and women: a monocentric study.

Luigi Maione1,2,3, Giovanna Pala2,4,5, Claire Bouvattier1,4,3, Séverine Trabado1,3,6, Georgios Papadakis2,7, Philippe Chanson1,2,3, Jérôme Bouligand1,3,6, Nelly Pitteloud7, Andrew A Dwyer8, Mohamad Maghnie5, Jacques Young1,2,3.   

Abstract

CONTEXT: Congenital hypogonadotropic hypogonadism/Kallmann syndrome (CHH/KS) is a rare condition characterized by gonadotropin deficiency and pubertal failure. Adult height (AH) in patients with CHH/KS has not been well studied.
OBJECTIVE: To assess AH in a large cohort of patients with CHH/KS. PATIENTS: A total of 219 patients (165 males, 54 females). Parents and siblings were included.
METHODS: AH was assessed in patients and family members. AH was compared to the general French population, mid parental target height (TH) and between patients and same-sex siblings. Delta height (∆H) was considered as the difference between AH and parental TH. ∆H was compared between patients and siblings, normosmic CHH and KS (CHH with anosmia/hyposmia), and according to underlying genetic defect. We examined the correlations between ∆H and age at diagnosis and therapeutically induced individual statural gain.
RESULTS: Mean AH in men and women with CHH/KS was greater than that in the French general population. Patients of both sexes had AH > TH. Males with CHH/KS were significantly, albeit moderately, taller than their brothers. ∆H was higher in CHH/KS compared to unaffected siblings (+6.2 ± 7.2 cm vs +3.4 ± 5.2 cm, P < 0.0001). ∆H was positively correlated with age at diagnosis. Neither olfactory function (normosmic CHH vs KS) nor specific genetic cause impacted ∆H. Individual growth during replacement therapy inversely correlated with the age at initiation of hormonal treatment (P < 0.0001).
CONCLUSIONS: CHH/KS is associated with higher AH compared to the general population and mid-parental TH. Greater height in CHH/KS than siblings indicates that those differences are in part independent of an intergenerational effect.

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Year:  2020        PMID: 31770102     DOI: 10.1530/EJE-19-0537

Source DB:  PubMed          Journal:  Eur J Endocrinol        ISSN: 0804-4643            Impact factor:   6.664


  3 in total

1.  Novel Microdeletion in the X Chromosome Leads to Kallmann Syndrome, Ichthyosis, Obesity, and Strabismus.

Authors:  Wanlu Ma; Jiangfeng Mao; Xi Wang; Lian Duan; Yuwen Song; Xiaolan Lian; Junjie Zheng; Zhaoxiang Liu; Min Nie; Xueyan Wu
Journal:  Front Genet       Date:  2020-06-24       Impact factor: 4.599

2.  GnRH Deficient Patients With Congenital Hypogonadotropic Hypogonadism: Novel Genetic Findings in ANOS1, RNF216, WDR11, FGFR1, CHD7, and POLR3A Genes in a Case Series and Review of the Literature.

Authors:  Vassos Neocleous; Pavlos Fanis; Meropi Toumba; George A Tanteles; Melpo Schiza; Feride Cinarli; Nicolas C Nicolaides; Anastasis Oulas; George M Spyrou; Christos S Mantzoros; Dimitrios Vlachakis; Nicos Skordis; Leonidas A Phylactou
Journal:  Front Endocrinol (Lausanne)       Date:  2020-08-28       Impact factor: 5.555

3.  Predictors of reproductive and non-reproductive outcomes of gonadotropin mediated pubertal induction in male patients with congenital hypogonadotropic hypogonadism (CHH).

Authors:  B Cangiano; G Goggi; S Federici; C Bresesti; L Cotellessa; F Guizzardi; V Vezzoli; P Duminuco; L Persani; M Bonomi
Journal:  J Endocrinol Invest       Date:  2021-03-18       Impact factor: 4.256

  3 in total

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