Literature DB >> 31759938

Effect of evolocumab on lipoprotein apheresis requirement and lipid levels: Results of the randomized, controlled, open-label DE LAVAL study.

Seth J Baum1, Tiziana Sampietro2, Dev Datta3, Patrick M Moriarty4, Beat Knusel5, Jingjing Schneider5, Ransi Somaratne5, Christopher Kurtz5, Bernd Hohenstein6.   

Abstract

BACKGROUND: Lipoprotein apheresis (LA) can effectively lower lipoproteins but is an invasive procedure.
OBJECTIVE: The objective of this study was to evaluate whether evolocumab can reduce LA requirement in patients undergoing chronic LA.
METHODS: Patients on regular weekly or every-2-week LA and moderate- to high-intensity statin (if tolerated) with pre-LA low-density lipoprotein cholesterol (LDL-C) levels ≥2.6 mmol/L (100 mg/dL) to ≤4.9 mmol/L (190 mg/dL) were randomized to continue the same LA frequency, or discontinue LA and receive evolocumab 140 mg every-2-weeks subcutaneously for 6 weeks. At week 6, all patients received only open-label evolocumab for 18 weeks. The primary endpoint was LA avoidance at the end of 6 weeks based on achieving pre-LA LDL-C <2.6 mmol/L at week 4.
RESULTS: Thirty-nine patients (mean [SD] age 62 [10] years, 59% male, 82% with familial hypercholesterolemia) were randomized (evolocumab, n = 19; LA, n = 20). At the end of 6 weeks, more patients receiving evolocumab avoided LA than those receiving LA (84% vs 10%; treatment difference, 74% [95% CI: 45, 87]; P < .0001). Thirty patients (77%) did not require LA at 24 weeks. Evolocumab reduced pre-LA LDL-C by 50% from the baseline to week 4 compared with a 3% increase in the LA arm. Pre-LA LDL-C <1.8 mmol/L (70 mg/dL) was achieved by 10 patients (53%) receiving evolocumab and none receiving LA (week 4). Safety was comparable between arms.
CONCLUSION: Evolocumab treatment significantly reduced LA requirement in patients undergoing chronic LA. In addition, >50% of patients achieved LDL-C <1.8 mmol/L on evolocumab alone, demonstrating that in patients with pre-LA LDL-C ≤4.9 mmol/L, evolocumab may replace LA.
Copyright © 2019 National Lipid Association. Published by Elsevier Inc. All rights reserved.

Entities:  

Keywords:  Cardiovascular disease; Heterozygous familial hypercholesterolemia; Hypercholesterolemia; Lipoprotein apheresis; Monoclonal antibody; PCSK9

Mesh:

Substances:

Year:  2019        PMID: 31759938     DOI: 10.1016/j.jacl.2019.10.003

Source DB:  PubMed          Journal:  J Clin Lipidol        ISSN: 1876-4789            Impact factor:   4.766


  4 in total

1.  Preclinical development and phase 1 trial of a novel siRNA targeting lipoprotein(a).

Authors:  Michael J Koren; Monica Florio; Patrick Maurice Moriarty; Seth J Baum; Joel Neutel; Martha Hernandez-Illas; Howard S Weintraub; Helina Kassahun; Stacey Melquist; Tracy Varrieur; Saptarsi M Haldar; Winnie Sohn; Huei Wang; Mary Elliott-Davey; Brooke M Rock; Tao Pei; Oliver Homann; Jennifer Hellawell; Gerald F Watts
Journal:  Nat Med       Date:  2022-01-13       Impact factor: 87.241

Review 2.  Lipoprotein(a) Lowering-From Lipoprotein Apheresis to Antisense Oligonucleotide Approach.

Authors:  Maria Francesca Greco; Cesare R Sirtori; Alberto Corsini; Marat Ezhov; Tiziana Sampietro; Massimiliano Ruscica
Journal:  J Clin Med       Date:  2020-07-03       Impact factor: 4.964

Review 3.  Efficacy and safety of PCSK9 monoclonal antibodies: an evidence-based review and update.

Authors:  Rasha Kaddoura; Bassant Orabi; Amar M Salam
Journal:  J Drug Assess       Date:  2020-08-11

4.  Maculopapular Exanthema After the Second Dose of Evolocumab.

Authors:  Victoria Ghernautan; Masoud Amini; Issac Sachmechi
Journal:  Cureus       Date:  2021-05-26
  4 in total

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