Literature DB >> 31744502

Correction to: Inhibition of the key metabolic pathways, glycolysis and lipogenesis, of oral cancer by bitter melon extract.

Subhayan Sur1, Hiroshi Nakanishi1, Colin Flaveny2, Joseph E Ippolito3, Jane McHowat1, David A Ford4, Ratna B Ray5.   

Abstract

Following publication of the original article [1], it was reported that Fig. 1c was not entirely readable due to overlapping Fig. 1d. The publishers apologise for this error.

Entities:  

Year:  2019        PMID: 31744502      PMCID: PMC6864944          DOI: 10.1186/s12964-019-0475-7

Source DB:  PubMed          Journal:  Cell Commun Signal        ISSN: 1478-811X            Impact factor:   5.712


Correction to: Cell Commun Signal https://doi.org/10.1186/s12964-019-0447-y Following publication of the original article [1], it was reported that Fig. 1c was not entirely readable due to overlapping Fig. 1d. The publishers apologise for this error.
Fig. 1

BME treatment reduces expression of glycolytic genes. a: Relative mRNA expression of GLUT-1, PFKP, PKM, LDHA, and PDK3 was analysed by q-RT-PCR in Cal27 and JHU022 cells with/without BME. 18 s gene was used as internal control. b: Cell lysates from Cal27 and JHU022 with or without BME treatment for 30 h were subjected to Western blot analysis for GLUT-1, PFKP, LDHA, PKM and PDK3 using specific antibodies. The membrane was reprobed with antibody to actin as an internal control. c: Quantitative of Western blot band intensities using Image-J software. Small bar indicates standard error (*, p < 0.05; **, p < 0.01; *** p < 0.001). d: Schematic diagram showing different genes regulate glycolysis and effect of BME on the genes

The updated Fig. 1 is supplied below. The original article [1] has been corrected. BME treatment reduces expression of glycolytic genes. a: Relative mRNA expression of GLUT-1, PFKP, PKM, LDHA, and PDK3 was analysed by q-RT-PCR in Cal27 and JHU022 cells with/without BME. 18 s gene was used as internal control. b: Cell lysates from Cal27 and JHU022 with or without BME treatment for 30 h were subjected to Western blot analysis for GLUT-1, PFKP, LDHA, PKM and PDK3 using specific antibodies. The membrane was reprobed with antibody to actin as an internal control. c: Quantitative of Western blot band intensities using Image-J software. Small bar indicates standard error (*, p < 0.05; **, p < 0.01; *** p < 0.001). d: Schematic diagram showing different genes regulate glycolysis and effect of BME on the genes
  1 in total

1.  Inhibition of the key metabolic pathways, glycolysis and lipogenesis, of oral cancer by bitter melon extract.

Authors:  Subhayan Sur; Hiroshi Nakanishi; Colin Flaveny; Joseph E Ippolito; Jane McHowat; David A Ford; Ratna B Ray
Journal:  Cell Commun Signal       Date:  2019-10-21       Impact factor: 5.712

  1 in total
  1 in total

1.  Molecular alterations in oral cancer using high-throughput proteomic analysis of formalin-fixed paraffin-embedded tissue.

Authors:  Varshasnata Mohanty; Yashwanth Subbannayya; Shankargouda Patil; Vinuth N Puttamallesh; Mohd Altaf Najar; Keshava K Datta; Sneha M Pinto; Sameera Begum; Neeta Mohanty; Samapika Routray; Riaz Abdulla; Jay Gopal Ray; David Sidransky; Harsha Gowda; T S Keshava Prasad; Aditi Chatterjee
Journal:  J Cell Commun Signal       Date:  2021-03-08       Impact factor: 5.782

  1 in total

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