Literature DB >> 31743304

Lack of an Exposure Response and Interaction With HLA-DPβ1 and DRβ1 Polymorphisms in the Development of Beryllium Toxicity in a High Beryllium Exposure Cohort.

Vitri Widyaningsih1, Kenneth Rosenman, Mary Jo Reilly, Ling Wang, Dorothy R Pathak, Joseph C Gardiner, Carol Rice, Dimitri Monos, Milton Rossman.   

Abstract

OBJECTIVE: To evaluate interaction of HLA-DPβ1 and DRβ1 polymorphisms with metrics of beryllium exposure, in the development of beryllium sensitization (BeS) and chronic beryllium disease (CBD).
METHODS: A matched case-control study of 61 CBD, 41 BeS, and 259 controls from two beryllium-processing facilities.
RESULTS: BES and CBD were significantly associated with presence of DPβE69. Dose response of exposure was not observed for the development of BES and CBD with/without adjustment for DPβE69 (P > 0.05). The DRβE71 polymorphism was more common in BeS than CBD after adjusting for exposure and maybe a protective factor (aOR 0.4, 95% CI 0.2 to 0.9) against the progression of BeS to CBD.
CONCLUSION: No exposure-response association was found, which may reflect that the workers in this high exposure cohort were above a threshold level where an exposure-response could be observed.

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Year:  2020        PMID: 31743304     DOI: 10.1097/JOM.0000000000001766

Source DB:  PubMed          Journal:  J Occup Environ Med        ISSN: 1076-2752            Impact factor:   2.162


  1 in total

1.  HLA-DPB1 E69 genotype and exposure in beryllium sensitisation and disease.

Authors:  James Crooks; Margaret M Mroz; Michael VanDyke; Alison McGrath; Christine Schuler; Erin C McCanlies; M Abbas Virji; Kenneth D Rosenman; Milton Rossman; Carol Rice; Dimitri Monos; Tasha E Fingerlin; Lisa A Maier
Journal:  Occup Environ Med       Date:  2021-09-17       Impact factor: 4.402

  1 in total

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