Literature DB >> 31606152

Clinical and Molecular Disease Spectrum and Outcomes in Patients with Infantile-Onset Pompe Disease.

Neerja Gupta1, Zoheb B Kazi2, Sheela Nampoothiri3, Sujatha Jagdeesh4, Madhulika Kabra5, Ratna Dua Puri6, Mamta Muranjan7, Mani Kalaivani8, Catherine Rehder2, Deeksha Bali2, Ishwar C Verma6, Priya S Kishnani2.   

Abstract

OBJECTIVES: To evaluate the clinical and molecular spectrum, and factors affecting clinical outcome of patients in India diagnosed with infantile-onset Pompe disease (IOPD). STUDY
DESIGN: In this multicenter, cross-sectional study, we evaluated the records of 77 patients with IOPD to analyze their clinical course, outcomes, and factors influencing the outcomes.
RESULTS: Of the 77 patients with IOPD, phenotype data were available in 59; 46 (78%) had the classic phenotype. Overall, 58 of 77 (75%) and 19 of 77 (25%) patients were symptomatic before and after age 6 months, respectively. Alpha-glucosidase gene variant analysis available for 48 patients (96 alleles) showed missense variants in 49 alleles. Cross-reactive immunologic material (CRIM) status could be determined or predicted in 44 of 48 patients. In total, 32 of 44 patients (72%) were CRIM-positive, and 12 of 44 patients (27%) were CRIM-negative. Thirty-nine cases received enzyme-replacement therapy (ERT), alglucosidase alfa, and 38 patients never received ERT. Median age at initiation of ERT was 6.5 months. Response to ERT was better in babies who had CRIM-positive, non-classic IOPD.
CONCLUSIONS: This study highlights the clinical spectrum of IOPD in India and provides an insight on various factors, such as undernutrition, feeding difficulties, and recurrent respiratory infection, as possible factors influencing clinical outcomes in these patients. The study also reiterates the importance of raising awareness among clinicians about the need for early diagnosis and timely treatment of IOPD.
Copyright © 2019 Elsevier Inc. All rights reserved.

Entities:  

Keywords:  GAA variants; classic; cross-reactive immunologic material; enzyme-replacement therapy; infantile-onset Pompe disease; non-classic

Year:  2019        PMID: 31606152     DOI: 10.1016/j.jpeds.2019.08.058

Source DB:  PubMed          Journal:  J Pediatr        ISSN: 0022-3476            Impact factor:   4.406


  5 in total

Review 1.  Congenital or Early Developing Neuromuscular Diseases Affecting Feeding, Swallowing and Speech - A Review of the Literature from January 1998 to August 2021.

Authors:  Lotta Sjögreen; Lisa Bengtsson
Journal:  J Neuromuscul Dis       Date:  2022

2.  Phenotypic implications of pathogenic variant types in Pompe disease.

Authors:  Manuel A Viamonte; Stephanie L Filipp; Zara Zaidi; Matthew J Gurka; Barry J Byrne; Peter B Kang
Journal:  J Hum Genet       Date:  2021-05-11       Impact factor: 3.172

3.  NGS-based expanded carrier screening for genetic disorders in North Indian population reveals unexpected results - a pilot study.

Authors:  Kanika Singh; Sunita Bijarnia-Mahay; V L Ramprasad; Ratna Dua Puri; Sandhya Nair; Sheetal Sharda; Renu Saxena; Sudha Kohli; Samarth Kulshreshtha; Indrani Ganguli; Kanwal Gujral; Ishwar C Verma
Journal:  BMC Med Genet       Date:  2020-11-02       Impact factor: 2.103

4.  Three-dimensional tissue-engineered human skeletal muscle model of Pompe disease.

Authors:  Jason Wang; Chris J Zhou; Alastair Khodabukus; Sabrina Tran; Sang-Oh Han; Aaron L Carlson; Lauran Madden; Priya S Kishnani; Dwight D Koeberl; Nenad Bursac
Journal:  Commun Biol       Date:  2021-05-05

5.  Infantile-onset Pompe disease complicated by sickle cell anemia: Case report and management considerations.

Authors:  Rodrigo Tzovenos Starosta; Ying-Chen Claire Hou; Katelyn Leestma; Prapti Singh; Luke Viehl; Linda Manwaring; Jorge Luis Granadillo; Molly C Schroeder; Jamie N Colombo; Halana Whitehead; Patricia Irene Dickson; Monica L Hulbert; Hoanh Thi Nguyen
Journal:  Front Pediatr       Date:  2022-09-28       Impact factor: 3.569

  5 in total

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