Literature DB >> 3156763

Increase of alcohol dehydrogenase and protein content of liver following chronic ethanol administration.

L Buris, G Csabai, M Fodor, M Varga.   

Abstract

Increased alcohol tolerance following chronic alcohol administration has been explained by increased mitochondrial oxidation of NADH and or increased activation of MEOS. According to our experiments this increased tolerance after chronic alcohol consumption is connected with an increased activity of ADH.

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Year:  1985        PMID： 3156763     DOI： 10.1016/0014-5793(85)80972-8

Source DB:  PubMed          Journal:  FEBS Lett        ISSN： 0014-5793            Impact factor:   4.124


  3 in total

1.  Gene-selective histone H3 acetylation in the absence of increase in global histone acetylation in liver of rats chronically fed alcohol.

Authors:  Pil-Hoon Park; Robert W Lim; Shivendra D Shukla
Journal:  Alcohol Alcohol       Date:  2012-02-02       Impact factor: 2.826

2.  Hepatic ethanol elimination kinetics in patients with cirrhosis.

Authors:  Gitte Dam; Michael Sørensen; Ole Lajord Munk; Susanne Keiding
Journal:  Scand J Gastroenterol       Date:  2009       Impact factor: 2.423

3.  Hepatic alcohol dehydrogenase activity in alcoholic subjects with and without liver disease.

Authors:  F Vidal; J Perez; J Morancho; B Pinto; C Richart
Journal:  Gut       Date:  1990-06       Impact factor: 23.059
  3 in total

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