Literature DB >> 31563038

Effects of acute psychosocial stress on the hypothalamic-pituitary-thyroid (HPT) axis in healthy women.

Susanne Fischer1, Jana Strahler2, Charlotte Markert2, Nadine Skoluda3, Johanna M Doerr4, Mattes Kappert5, Urs M Nater3.   

Abstract

OBJECTIVE: The individual set point of the hypothalamic-pituitary-thyroid (HPT) axis is largely genetically determined. Apart from this genetic predisposition, the HPT axis may also be malleable to environmental demands such as psychosocial stress. Indeed, previous research has indicated that critical life events often precede the onset of autoimmune thyroid diseases, and subtle abnormalities in HPT functioning are present in some patients with stress-related disorders such as depression. However, no studies have investigated whether exposure to psychosocial stress leads to an immediate activation of the HPT axis.
METHODS: A total of N = 30 healthy women attended two laboratory appointments in a randomized order. An intravenous catheter was inserted at the beginning of each appointment. In the stress session, this was followed by the Trier Social Stress Test (TSST). Plasma samples to determine thyroid-stimulating hormone (TSH), triiodothyronine (T3), and thyroxine (T4) were taken at baseline and 20, 50, and 110 min after the TSST started. In the control session, participants rested and were instructed to read magazines, while the sampling schedule was maintained.
RESULTS: There was a significant rise in TSH concentrations in response to the TSST, with a peak observed 20 min after stressor onset, and a steady decline thereafter. No such response was observed in the control session. The TSST did not increase T3 or T4.
CONCLUSION: The finding that acute psychosocial stress is able to elicit a significant increase in TSH is relevant to our understanding of a number of stress-related illnesses presenting with abnormalities of the HPT axis.
Copyright © 2019 Elsevier Ltd. All rights reserved.

Entities:  

Keywords:  Psychosocial; Stress; Thyroid-Stimulating hormone; Thyroxine; Triiodothyronine

Year:  2019        PMID: 31563038     DOI: 10.1016/j.psyneuen.2019.104438

Source DB:  PubMed          Journal:  Psychoneuroendocrinology        ISSN: 0306-4530            Impact factor:   4.905


  5 in total

1.  Increased prevalence of subclinical hypothyroidism in female hospitalized patients with depression.

Authors:  Shuai Zhao; Zhilu Chen; Xumiao Wang; Zhijian Yao; Qing Lu
Journal:  Endocrine       Date:  2020-10-16       Impact factor: 3.633

2.  A prospective, observational clinical trial on the impact of COVID-19-related national lockdown on thyroid hormone in young males.

Authors:  Giulia Brigante; Giorgia Spaggiari; Barbara Rossi; Antonio Granata; Manuela Simoni; Daniele Santi
Journal:  Sci Rep       Date:  2021-03-29       Impact factor: 4.379

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Authors:  Dong Weiwei; Wu Bei; Wang Hong; Wu Cailan; Shao Hailin; Xu Donghong; Wang Xiaolai; Hao Zhaohu; Li Shijun; Tan Jian; Jia Qiang
Journal:  Int J Endocrinol       Date:  2022-01-07       Impact factor: 2.803

4.  The hypothalamic-pituitary-adrenal and -thyroid axes activation lasting one year after an earthquake swarm: results from a big data analysis.

Authors:  G Spaggiari; M Setti; S Tagliavini; L Roli; M C De Santis; T Trenti; V Rochira; D Santi
Journal:  J Endocrinol Invest       Date:  2020-10-29       Impact factor: 4.256

5.  Development of a visual tool to assess six dimensions of health and its validation in patients with endocrine disorders.

Authors:  Christian Fazekas; Dennis Linder; Franziska Matzer; Christian Vajda; Alexander Avian; Verena Theiler-Schwetz; Christian Trummer; Julia Došen; Jelena Rokvic; Marco Mohl; Stefan Pilz
Journal:  Wien Klin Wochenschr       Date:  2021-02-04       Impact factor: 2.275

  5 in total

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