| Literature DB >> 31543148 |
Brian Czaya1, Christian Faul2.
Abstract
High serum concentrations of the phosphaturic hormone, fibroblast growth factor 23 (FGF23), contribute to various tissue injuries. In chronic kidney disease, the sources of FGF23 and the stimuli that control FGF23 production differ from those in the physiologic scenario. Mediators of inflammation are intensively studied as potential factors that contribute to FGF23 elevations and thereby might function as drug targets to lower FGF23 levels. The present study focuses on tumor necrosis factor.Entities:
Mesh:
Substances:
Year: 2019 PMID: 31543148 DOI: 10.1016/j.kint.2019.05.018
Source DB: PubMed Journal: Kidney Int ISSN: 0085-2538 Impact factor: 10.612