Literature DB >> 31496163

[Correlation of phosphorylated S6 protein expression in blood and brain tissue in mice and rats with kainic acid-induced seizure].

Miao Fan1, Shuminin Dong1, Xinyi Zou1, Boyuan Zheng1, Yurun Huang1, Jianda Wang2, Linghui Zeng1.   

Abstract

OBJECTIVE: To determine the correlation of phosphorylated ribosomal S6 protein (P-S6) content in blood and brain tissue in mice and rats with seizure.
METHODS: Seizure models were induced by intraperitoric injection of kainic acid (KA) in C57BL/mice and SD rats. Flow cytometry was used to detect the content of P-S6 in blood; Western blot was used to detect the expression of P-S6 in brain tissues. The correlation between P-S6 expression in blood and in brain tissue was examine by Pearson analysis, and the correlation between P-S6 expression in blood and the severity of seizure was also observed.
RESULTS: Western blotting analysis showed that the expression of P-S6 was significantly increased in peripheral blood and brain tissue in mice 1 h after KA-induced seizure,and the expression levels increased to (1.49±0.45) times (P<0.05) and (2.55±0.66) times (P <0.01) of the control group, respectively. Flow cytometry showed that the positive percentage and average fluorescence intensity of P-S6 in the blood of mice increased significantly 1 h after KA-induced seizures (P<0.01), which was consistent with the expression of P-S6 in brain tissue (r=0.8474, P<0.01). Flow cytometry showed that the average fluorescence intensity of P-S6 in blood increased from 14.89±9.75 to 52.35±21.72 (P<0.01) in rats with seizure, which was consistent with the change of P-S6 in brain tissue (r=0.9385, P<0.01). Rats with higher levels of seizure were of higher levels of P-S6 in peripheral blood.
CONCLUSIONS: Consistent correlation of P-S6 expression is demonstrated in peripheral blood and in brain tissue after KA-induced seizure, suggesting that the expression of P-S6 in blood can accurately reflect the changes of mTOR signaling pathway in brain tissue.

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Year:  2019        PMID: 31496163      PMCID: PMC8800752          DOI: 10.3785/j.issn.1008-9292.2019.06.11

Source DB:  PubMed          Journal:  Zhejiang Da Xue Xue Bao Yi Xue Ban        ISSN: 1008-9292


  21 in total

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2.  De novo somatic mutations in components of the PI3K-AKT3-mTOR pathway cause hemimegalencephaly.

Authors:  Jeong Ho Lee; My Huynh; Jennifer L Silhavy; Sangwoo Kim; Tracy Dixon-Salazar; Andrew Heiberg; Eric Scott; Vineet Bafna; Kiley J Hill; Adrienne Collazo; Vincent Funari; Carsten Russ; Stacey B Gabriel; Gary W Mathern; Joseph G Gleeson
Journal:  Nat Genet       Date:  2012-06-24       Impact factor: 38.330

3.  BRAF V600E mutation in epilepsy-associated glioneuronal tumors: Prevalence and correlation with clinical features in a Chinese population.

Authors:  Yin-Xi Zhang; Chun-Hong Shen; Yi Guo; Yang Zheng; Jun-Ming Zhu; Yao Ding; Ye-Lei Tang; Shuang Wang; Mei-Ping Ding
Journal:  Seizure       Date:  2016-12-09       Impact factor: 3.184

4.  STRADalpha deficiency results in aberrant mTORC1 signaling during corticogenesis in humans and mice.

Authors:  Ksenia A Orlova; Whitney E Parker; Gregory G Heuer; Victoria Tsai; Jason Yoon; Marianna Baybis; Robert S Fenning; Kevin Strauss; Peter B Crino
Journal:  J Clin Invest       Date:  2010-04-26       Impact factor: 14.808

5.  Tsc2 gene inactivation causes a more severe epilepsy phenotype than Tsc1 inactivation in a mouse model of tuberous sclerosis complex.

Authors:  Ling-Hui Zeng; Nicholas R Rensing; Bo Zhang; David H Gutmann; Michael J Gambello; Michael Wong
Journal:  Hum Mol Genet       Date:  2010-11-09       Impact factor: 6.150

6.  Activation of microglia/macrophages expressing phosphorylated S6 ribosomal protein in a case of hemimegalencephaly with progressive calcification and atrophy.

Authors:  Yutaka Nonoda; Yoshiaki Saito; Masayuki Itoh; Eiji Nakagawa; Kenji Sugai; Akio Takahashi; Taisuke Otsuki; Yuko Saito; Kunimasa Arima; Masashi Mizuguchi; Yu-ichi Goto; Masayuki Sasaki
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7.  Rapamycin reduces seizure frequency in tuberous sclerosis complex.

Authors:  Jennifer Muncy; Ian J Butler; Mary Kay Koenig
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Review 8.  mTOR pathway inhibition as a new therapeutic strategy in epilepsy and epileptogenesis.

Authors:  Rita Citraro; Antonio Leo; Andrew Constanti; Emilio Russo; Giovambattista De Sarro
Journal:  Pharmacol Res       Date:  2016-04-02       Impact factor: 7.658

9.  Molecular isoforms of high-mobility group box 1 are mechanistic biomarkers for epilepsy.

Authors:  Lauren Elizabeth Walker; Federica Frigerio; Teresa Ravizza; Emanuele Ricci; Karen Tse; Rosalind E Jenkins; Graeme John Sills; Andrea Jorgensen; Luca Porcu; Thimmasettappa Thippeswamy; Tiina Alapirtti; Jukka Peltola; Martin J Brodie; Brian Kevin Park; Anthony Guy Marson; Daniel James Antoine; Annamaria Vezzani; Munir Pirmohamed
Journal:  J Clin Invest       Date:  2017-05-15       Impact factor: 14.808

Review 10.  Roles of mTOR Signaling in Brain Development.

Authors:  Da Yong Lee
Journal:  Exp Neurobiol       Date:  2015-09-17       Impact factor: 3.261

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