| Literature DB >> 31484534 |
Bhuwan Giri1, Vineet K Gupta1, Brianna Yaffe1, Shrey Modi1, Pooja Roy1, Vrishketan Sethi1, Shweta P Lavania1, Selwyn M Vickers1, Vikas Dudeja1, Sulagna Banerjee1, Justin Watts1, Ashok Saluja2.
Abstract
Following publication of the original article [1], the authors found an error in Figure 3. The middle panel of Figure 3a was inadvertently duplicated.Entities:
Year: 2019 PMID: 31484534 PMCID: PMC6727517 DOI: 10.1186/s12967-019-2048-3
Source DB: PubMed Journal: J Transl Med ISSN: 1479-5876 Impact factor: 5.531
Fig. 3a Treatment with triptolide resulted in a decrease in colony forming ability of AML cell lines THP-1 and KG1a as measured by a methylcellulose based colony forming assay. Treatment with triptolide also caused a decrease in surface markers of commonly expressed stem cell markers in AML cell lines THP-1 (b) and KG1a (c). n = 3, *p < 0.05 when compared to untreated cells, data shows mean ± SD
Fig. 3a Treatment with triptolide resulted in a decrease in colony forming ability of AML cell lines THP-1 and KG1a as measured by a methylcellulose based colony forming assay. Treatment with triptolide also caused a decrease in surface markers of commonly expressed stem cell markers in AML cell lines THP-1 (b) and KG1a (c). n = 3, *p < 0.05 when compared to untreated cells, data shows mean ± SD