Literature DB >> 3146301

Occurrence of holoprosencephaly in chromosome 13 disorders cannot be explained by duplication/deficiency of a single locus.

G N Wilson1, M Dasouki, M Barr.   

Abstract

Four cases of holoprosencephaly with duplication/deletion involving chromosome 13 are presented and additional cases are summarized from the literature. When examined as a series, the duplications (trisomy 13, trisomy 13pter----q14) and deletions (deletion 13q12----qter, deletion 13q31----qter, ring 13 with deletion 13q14----qter) exclude deletion or duplication of single chromosome 13 bands as the cause for holoprosencephaly. Increased dosage of the 13pter----q14 region relative to the 13q14----qter region as the cause is also ruled out by the duplication 13q21----qter cases reported in the literature. Altered timing of forebrain development, causing reversion to a more primitive embryonic and phylogenetic brain structure, is related to dosage imbalance of at least two chromosome 13 regions.

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Year:  1986        PMID: 3146301     DOI: 10.1002/ajmg.1320250610

Source DB:  PubMed          Journal:  Am J Med Genet Suppl        ISSN: 1040-3787


  2 in total

Review 1.  13q13.1-q13.2 deletion in tetralogy of Fallot: clinical report and a literature review.

Authors:  Gregory Costain; Candice K Silversides; Christian R Marshall; Mary Shago; Nicholas Costain; Anne S Bassett
Journal:  Int J Cardiol       Date:  2010-07-03       Impact factor: 4.164

2.  Overexpression of esterase D in kidney from trisomy 13 fetuses.

Authors:  S Loughna; P Bennett; G Gau; K Nicolaides; S Blunt; G Moore
Journal:  Am J Hum Genet       Date:  1993-10       Impact factor: 11.025

  2 in total

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