| Literature DB >> 31439758 |
Yang Liu1,2, Yuling You1, Zhike Lu3, Jiang Yang1, Panpan Li1, Lun Liu1, Henan Xu1, Yamei Niu4, Xuetao Cao5,2,6.
Abstract
Host cell metabolism can be modulated by viral infection, affecting viral survival or clearance. Yet the cellular metabolism rewiring mediated by the N 6-methyladenosine (m6A) modification in interactions between virus and host remains largely unknown. Here we report that in response to viral infection, host cells impair the enzymatic activity of the RNA m6A demethylase ALKBH5. This behavior increases the m6A methylation on α-ketoglutarate dehydrogenase (OGDH) messenger RNA (mRNA) to reduce its mRNA stability and protein expression. Reduced OGDH decreases the production of the metabolite itaconate that is required for viral replication. With reduced OGDH and itaconate production in vivo, Alkbh5-deficient mice display innate immune response-independent resistance to viral exposure. Our findings reveal that m6A RNA modification-mediated down-regulation of the OGDH-itaconate pathway reprograms cellular metabolism to inhibit viral replication, proposing potential targets for controlling viral infection.Entities:
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Year: 2019 PMID: 31439758 DOI: 10.1126/science.aax4468
Source DB: PubMed Journal: Science ISSN: 0036-8075 Impact factor: 47.728